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Items: 4

1.

The histone H3G34R mutation disrupts the epigenome via catalytic inactivation of the ASH1 H3K36 methyltransferase

(Submitter supplied) This SuperSeries is composed of the SubSeries listed below.
Organism:
Neurospora crassa
Type:
Expression profiling by high throughput sequencing; Genome binding/occupancy profiling by high throughput sequencing; Methylation profiling by high throughput sequencing
Platforms:
GPL23150 GPL20660 GPL16164
20 Samples
Download data: BIGWIG, IGV, TAB, TDF, XLSX
Series
Accession:
GSE134452
ID:
200134452
2.

The histone H3G34R mutation disrupts the epigenome via catalytic inactivation of the ASH1 H3K36 methyltransferase [ChIP-seq]

(Submitter supplied) The recurrent mutation of histone variant H3.3 at glycine-34 (H3.3G34) defines a type of pediatric glioma. Characteristic changes to the epigenome associated with the disease are thought to be the consequence of altered methylation of the adjacent lysine-36 (K36) residue, but the complexity of this regulatory pathway in humans, combined with a multi-component disease etiology, has limited our understanding of how H3.3G34 mutations contribute to oncogenesis. more...
Organism:
Neurospora crassa
Type:
Genome binding/occupancy profiling by high throughput sequencing
Platforms:
GPL20660 GPL23150
12 Samples
Download data: BIGWIG, TDF
Series
Accession:
GSE134449
ID:
200134449
3.

Illumina HiSeq 4000 (Neurospora crassa)

Organism:
Neurospora crassa
22 Series
247 Samples
Download data
Platform
Accession:
GPL23150
ID:
100023150
4.

set-2_H3K9me3_ChIP-seq

Organism:
Neurospora crassa
Source name:
germinated conidia
Platform:
GPL23150
Series:
GSE134449 GSE134452
Download data: BIGWIG
Sample
Accession:
GSM3948560
ID:
303948560
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