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Genomic DNA copy number alterations of mouse B-cell lymphomas induced by ionizing radiation
PubMed Similar studies Analyze with GEO2R
Infection exposure is a causal factor in B-precursor acute lymphoblastic leukemia as a result of Pax5 inherited susceptibility
PubMed Full text in PMC Similar studies Analyze with GEO2R
Inducible PAX5 expression in a human B lymphoblastic leukemia cell line
PubMed Full text in PMC Similar studies Analyze with GEO2RSRA Run Selector
Pax5 restoration in a mouse model of B progenitor acute lymphoblastic leukemia
PubMed Full text in PMC Similar studies SRA Run Selector
Expression profiling of mouse bone marrow pre-B cells
Compare proB cells from WT, Tp53-/-, Lnk-/-, Tp53-/-Lnk-/- mice and Tp53-/-Lnk-/-B-ALL
Ebf1 or Pax5 Haploinsufficiency Synergizes with STAT5 Activation to Initiate Acute Lymphoblastic Leukemia
Pax5 is a tumor suppressor in mouse mutagenesis models of acute lymphoblastic leukemia
Identification of the dismal subtype of B-ALL with dysregulation of CDX2 and UBTF
Genome-wide signatures of differential DNA methylation in pediatric acute lymphoblastic leukemia
PubMed Full text in PMC Similar studies
Identification of mutations that cooperate with defects in B cell transcription factors to initiate leukemia
Identification of mutations that cooperate with defects in B cell transcription factors to initiate leukemia [RNA-seq]
Identification of mutations that cooperate with defects in B cell transcription factors to initiate leukemia [variation]
The gut microbiome protects genetically predisposed mice against leukemia
Molecular role of the PAX5-ETV6 oncoprotein in promoting B cell acute lymphoblastic leukemia
Antagonism of B cell enhancer networks by STAT5 drives leukemia and poor patient survival
The PAX5-JAK2 translocation acts as a dual-hit mutation that promotes aggressive B-cell leukemia via nuclear STAT5 activation
Germline Aberrations of PAX5 Cause Susceptibility to pre-B cell Acute Lymphoblastic Leukemia
Inhibition of Inflammatory Signaling in Pax5 Mutant Cells Mitigates B-cell leukemogenesis against leukemia
Assessment of ability of hTSLP to maintain primary CRLF2 B-ALL cells in a xenograft model in a state more similar to the parent leukemia
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