| The functional role of CST1 and CCL26 in asthma development. | The functional role of CST1 and CCL26 in asthma development.
Hoyer A, Chakraborty S, Lilienthal I, Konradsen JR, Katayama S, Söderhäll C., Free PMC Article | 02/27/2024 |
| Th2 cytokine signaling through IL-4Ralpha increases eotaxin-3 secretion and tension in human esophageal smooth muscle. | Th2 cytokine signaling through IL-4Rα increases eotaxin-3 secretion and tension in human esophageal smooth muscle.
Nelson MR, Zhang X, Podgaetz E, Wang X, Zhang Q, Pan Z, Spechler SJ, Souza RF. | 12/28/2023 |
| MCP-4 and Eotaxin-3 Are Novel Biomarkers for Chronic Obstructive Pulmonary Disease. | MCP-4 and Eotaxin-3 Are Novel Biomarkers for Chronic Obstructive Pulmonary Disease.
Fang C, Kang B, Zhao P, Ran J, Wang L, Zhao L, Luo H, Tao L., Free PMC Article | 05/23/2023 |
| [Expression and predictive value of type inflammatory cytokines in nasal secretion in eosinophilic chronic rhinosinusitis with nasal polyps]. | [Expression and predictive value of type Ⅱ inflammatory cytokines in nasal secretion in eosinophilic chronic rhinosinusitis with nasal polyps].
Zhang Z, Ma F, Liu J, Xie L, Cao W, Zhang Y., Free PMC Article | 01/14/2023 |
| Eotaxin-2 and eotaxin-3 in malaria exposure and pregnancy. | Eotaxin-2 and eotaxin-3 in malaria exposure and pregnancy.
Mancebo-Pérez C, Vidal M, Aguilar R, Barrios D, Bardají A, Ome-Kaius M, Menéndez C, Rogerson SJ, Dobaño C, Moncunill G, Requena P., Free PMC Article | 12/10/2022 |
| CCL26 silence represses colon cancer by inhibiting the EMT signaling pathway. | CCL26 silence represses colon cancer by inhibiting the EMT signaling pathway.
Sun A, Li Y, Jiang X. | 11/26/2022 |
| Type-2 Cytokines Promote the Secretion of the Eosinophil-Attractant CCL26 by Intestinal Epithelial Cells in Food-Sensitized Patients. | Type-2 Cytokines Promote the Secretion of the Eosinophil-Attractant CCL26 by Intestinal Epithelial Cells in Food-Sensitized Patients.
Vaccaro J, Canziani KE, Guzmán L, Bernedo V, García M, Altamirano EM, Feregotti E, Curciarello R, Muglia CI, Docena GH., Free PMC Article | 07/23/2022 |
| Analysis of the signal cross talk via CCL26 in the tumor microenvironment in osteosarcoma. | Analysis of the signal cross talk via CCL26 in the tumor microenvironment in osteosarcoma.
Kawano M, Iwasaki T, Itonaga I, Kubota Y, Tanaka K, Tsumura H., Free PMC Article | 12/18/2021 |
| Hypoxia directed migration of human naive monocytes is associated with an attenuation of cytokine release: indications for a key role of CCL26. | Hypoxia directed migration of human naïve monocytes is associated with an attenuation of cytokine release: indications for a key role of CCL26.
Hummitzsch L, Berndt R, Kott M, Rusch R, Faendrich F, Gruenewald M, Steinfath M, Albrecht M, Zitta K., Free PMC Article | 08/21/2021 |
| Increased serum CCL26 level is a potential biomarker for the effectiveness of anti-PD1 antibodies in patients with advanced melanoma. | Increased serum CCL26 level is a potential biomarker for the effectiveness of anti-PD1 antibodies in patients with advanced melanoma.
Fujimura T, Tanita K, Ohuchi K, Sato Y, Lyu C, Kambayashi Y, Fujisawa Y, Tanaka R, Hashimoto A, Aiba S. | 08/7/2021 |
| CCL26 is upregulated by nab-paclitaxel in pancreatic cancer-associated fibroblasts and promotes PDAC invasiveness through activation of the PI3K/AKT/mTOR pathway. | CCL26 is upregulated by nab-paclitaxel in pancreatic cancer-associated fibroblasts and promotes PDAC invasiveness through activation of the PI3K/AKT/mTOR pathway.
Chen X, An Y, Zhang Y, Xu D, Chen T, Yang Y, Chen W, Wu D, Zhang X. | 07/17/2021 |
| Lymphadenopathy in IgG4-related disease: a phenotype of severe activity and poor prognosis, with eotaxin-3 as a new biomarker. | Lymphadenopathy in IgG4-related disease: a phenotype of severe activity and poor prognosis, with eotaxin-3 as a new biomarker.
Takanashi S, Kikuchi J, Sasaki T, Akiyama M, Yasuoka H, Yoshimoto K, Seki N, Sugahara K, Chiba K, Kaneko Y, Takeuchi T. | 04/24/2021 |
| Pathogenesis of Eosinophilic Esophagitis: A Comprehensive Review of the Genetic and Molecular Aspects. | Pathogenesis of Eosinophilic Esophagitis: A Comprehensive Review of the Genetic and Molecular Aspects.
Ryu S, Lee KH, Tizaoui K, Terrazzino S, Cargnin S, Effenberger M, Shin JI, Kronbichler A., Free PMC Article | 02/27/2021 |
| Adipose Tissue Mast Cells Promote Human Adipose Beiging in Response to Cold. | Adipose Tissue Mast Cells Promote Human Adipose Beiging in Response to Cold.
Finlin BS, Confides AL, Zhu B, Boulanger MC, Memetimin H, Taylor KW, Johnson ZR, Westgate PM, Dupont-Versteegden EE, Kern PA., Free PMC Article | 10/24/2020 |
| CCL26 displays antimicrobial activity against S. pneumoniae, S. aureus, Non-typeable H. influenzae, and P. aeruginosa. | Eotaxin-3 (CCL26) exerts innate host defense activities that are modulated by mast cell proteases.
Gela A, Kasetty G, Jovic S, Ekoff M, Nilsson G, Mörgelin M, Kjellström S, Pease JE, Schmidtchen A, Egesten A. | 07/27/2020 |
| A significant positive correlation was found between the mucosal eosinophil count and the plasma levels of eotaxin-3. | Eotaxin-3 as a Plasma Biomarker for Mucosal Eosinophil Infiltration in Chronic Rhinosinusitis.
Yamada T, Miyabe Y, Ueki S, Fujieda S, Tokunaga T, Sakashita M, Kato Y, Ninomiya T, Kawasaki Y, Suzuki S, Saito H., Free PMC Article | 01/4/2020 |
| Carriers of a mutative allele in a haplotype within the promoter of IL33 had a lower IL33 DNA-me level and CCL26 gene expression correlated with eosinophil count. | Methylation profiles of IL33 and CCL26 in bronchial epithelial cells are associated with asthma.
Larouche M, Gagné-Ouellet V, Boucher-Lafleur AM, Larose MC, Plante S, Madore AM, Laviolette M, Flamand N, Chakir J, Laprise C. | 08/3/2019 |
| The eotaxin family currently includes three members: eotaxin-1 (CCL11), eotaxin-2 (CCL24), and eotaxin-3 (CCL26), and each was identified based on its ability | An emerging role for eotaxins in neurodegenerative disease.
Huber AK, Giles DA, Segal BM, Irani DN. | 07/6/2019 |
| This study concludes that serum CCL11, CCL24 and CCL26 are upregulated in primary biliary cholangitis (PBC); CCL11 and CCL26 are associated with fibrotic progression of PBC, but CCL24 is not. | Association of CCL11, CCL24 and CCL26 with primary biliary cholangitis.
Lin F, Shi H, Liu D, Zhang Z, Luo W, Mao P, Zhong R, Liang Y, Yang Z. | 05/11/2019 |
| These findings suggest that CISH plays a key role in the eosinophilic inflammation associated with bronchial asthma by regulating IL-13-induced CCL26 production. | CISH is a negative regulator of IL-13-induced CCL26 production in lung fibroblasts.
Takeshima H, Horie M, Mikami Y, Makita K, Miyashita N, Matsuzaki H, Noguchi S, Urushiyama H, Hiraishi Y, Mitani A, Borok Z, Nagase T, Yamauchi Y. | 04/6/2019 |
| When LoVo-P and HT29-C cells were cocultured with TAMs, CCL26 binding to the CCR3 receptor enhanced the invasiveness of LoVo-P and HT29-C cells by mobilizing intracellular Ca(2+)of TAMs to increase the expression of IL6 and IL8. | CCL26 Participates in the PRL-3-Induced Promotion of Colorectal Cancer Invasion by Stimulating Tumor-Associated Macrophage Infiltration.
Lan Q, Lai W, Zeng Y, Liu L, Li S, Jin S, Zhang Y, Luo X, Xu H, Lin X, Chu Z. | 03/9/2019 |
| Based on the ADEPT study data set, we report that airway mucosal expression of CCL26 was a robust discriminator of type 2 inflammation from healthy nonatopic subjects. Furthermore, airway mucosal CCL26 expression was best identified by using a panel of clinical biomarkers, including Feno values, bEOS counts, and expression of 2 novel markers, sCCL17 and sCCL26 | Identification of airway mucosal type 2 inflammation by using clinical biomarkers in asthmatic patients.
Silkoff PE, Laviolette M, Singh D, FitzGerald JM, Kelsen S, Backer V, Porsbjerg CM, Girodet PO, Berger P, Kline JN, Chupp G, Susulic VS, Barnathan ES, Baribaud F, Loza MJ, Airways Disease Endotyping for Personalized Therapeutics (ADEPT) study investigators. | 09/23/2017 |
| hypoxia-inducible factors activate the transcription of chemokine ligand 26 in cancer cells to recruit chemokine receptor 1-expressing myeloid-derived suppressor cells to the primary tumor | Hypoxia induces myeloid-derived suppressor cell recruitment to hepatocellular carcinoma through chemokine (C-C motif) ligand 26.
Chiu DK, Xu IM, Lai RK, Tse AP, Wei LL, Koh HY, Li LL, Lee D, Lo RC, Wong CM, Ng IO, Wong CC. | 08/5/2017 |
| this study shows that expression of CCL26 is increased in eosinophilic myocarditis patients compared to chronic lymphocytic myocarditis patients | Macrophages and cardiac fibroblasts are the main producers of eotaxins and regulate eosinophil trafficking to the heart.
Diny NL, Hou X, Barin JG, Chen G, Talor MV, Schaub J, Russell SD, Klingel K, Rose NR, Čiháková D., Free PMC Article | 07/8/2017 |
| The reduction of circulating levels of MCP-4, eotaxin-3, and MIP-1beta could be one of the mechanisms by which bariatric surgery contributes to the reduction of cardiovascular risk in these patients. | Circulating Levels of the Adipokines Monocyte Chemotactic Protein-4 (MCP-4), Macrophage Inflammatory Protein-1β (MIP-1β), and Eotaxin-3 in Severe Obesity and Following Bariatric Surgery.
Gentili A, Zaibi MS, Alomar SY, De Vuono S, Ricci MA, Alaeddin A, Siepi D, Boni M, Vaudo G, Trayhurn P, Lupattelli G. | 03/18/2017 |