| DGUOK-AS1 promotes cervical squamous cell carcinoma progression by suppressing miR-499a-5p that targets SPRR1B in vitro. | DGUOK-AS1 promotes cervical squamous cell carcinoma progression by suppressing miR-499a-5p that targets SPRR1B in vitro.
Song Y, Pan H, Yang L, Fan Y, Zhang H, Pan M, Zhang Y. | 01/8/2022 |
| Identification of small proline-rich protein 1B (SPRR1B) as a prognostically predictive biomarker for lung adenocarcinoma by integrative bioinformatic analysis. | Identification of small proline-rich protein 1B (SPRR1B) as a prognostically predictive biomarker for lung adenocarcinoma by integrative bioinformatic analysis.
Zhang Z, Shi R, Xu S, Li Y, Zhang H, Liu M, Zhu G, Chen C, Pan Z, Liu H, Chen J., Free PMC Article | 11/22/2021 |
| Identification of oral squamous cell carcinoma markers MUC2 and SPRR1B downstream of TANGO. | Identification of oral squamous cell carcinoma markers MUC2 and SPRR1B downstream of TANGO.
Sasahira T, Kurihara-Shimomura M, Shimomura H, Bosserhoff AK, Kirita T. | 05/29/2021 |
| Expression of SPRR1 and of SPRR2A is increased in mucosal samples from patients with chronic rhinosinusitis and appeared as a downstream result of TNF alpha modulation, which possibly resulted in epithelial barrier dysfunction. | RNA sequencing and pathway analysis identify tumor necrosis factor alpha driven small proline-rich protein dysregulation in chronic rhinosinusitis.
Ramakrishnan VR, Gonzalez JR, Cooper SE, Barham HP, Anderson CB, Larson ED, Cool CD, Diller JD, Jones K, Kinnamon SC., Free PMC Article | 05/19/2018 |
| Levels SPRR1B proteins were significantly higher in AD skin compared with the levels of these proteins in the skin of healthy subjects. | Expression of Cornified Envelope Proteins in Skin and Its Relationship with Atopic Dermatitis Phenotype.
Trzeciak M, Sakowicz-Burkiewicz M, Wesserling M, Dobaczewska D, Gleń J, Nowicki R, Pawelczyk T. | 01/28/2017 |
| expression of SPRR1B is upregulated in oral CSCs/CICs and SPRR1B has a role in cell growth by suppression of RASSF4 | Small proline-rich protein-1B is overexpressed in human oral squamous cell cancer stem-like cells and is related to their growth through activation of MAP kinase signal.
Michifuri Y, Hirohashi Y, Torigoe T, Miyazaki A, Fujino J, Tamura Y, Tsukahara T, Kanaseki T, Kobayashi J, Sasaki T, Takahashi A, Nakamori K, Yamaguchi A, Hiratsuka H, Sato N. | 11/16/2013 |
| Association of psoriasis to PGLYRP and SPRR genes at PSORS4 locus on 1q shows heterogeneity between Finnish, Swedish and Irish families. | Association of psoriasis to PGLYRP and SPRR genes at PSORS4 locus on 1q shows heterogeneity between Finnish, Swedish and Irish families.
Kainu K, Kivinen K, Zucchelli M, Suomela S, Kere J, Inerot A, Baker BS, Powles AV, Fry L, Samuelsson L, Saarialho-Kere U. | 05/7/2010 |
| Observational study of gene-disease association. (HuGE Navigator) | Variation in genes of the epidermal differentiation complex in German atopic dermatitis patients.
Stemmler S, Nothnagel M, Parwez Q, Petrasch-Parwez E, Epplen JT, Hoffjan S. | 09/16/2009 |
| SPRR1B is a valid biomarker for the study of the molecular mechanisms of squamous metaplasia. There is a definitive link between inflammation and squamous metaplasia in autoimmune-mediated dry eye disease | Small proline-rich protein 1B (SPRR1B) is a biomarker for squamous metaplasia in dry eye disease.
Li S, Nikulina K, DeVoss J, Wu AJ, Strauss EC, Anderson MS, McNamara NA., Free PMC Article | 01/21/2010 |
| SPRR1B expression pattern in nonsquamous tumors; findings indicate SPRR1B likely coupled to signals responsible for withdrawal from the proliferative state; overexpression for prolonged periods may disrupt normal progression of mitosis | SPRR1B overexpression enhances entry of cells into the G0 phase of the cell cycle.
Tesfaigzi Y, Wright PS, Belinsky SA. | 01/21/2010 |
| BMK1 (ERK5) regulates squamous differentiation marker SPRR1B transcription in Clara-like H441 cells. | BMK1 (ERK5) regulates squamous differentiation marker SPRR1B transcription in Clara-like H441 cells.
Reddy SP, Adiseshaiah P, Shapiro P, Vuong H. | 01/21/2010 |