An RNA binding region in CTCF regulates chromatin looping and CTCF nuclear organization

Comparative transcriptomics between species attributes reactogenicity pathways induced by the capsular group B meningococcal vaccine, 4CMenB, to the membrane-bound endotoxin of its outer membrane vesicle component.

DOT1L inhibition reveals a distinct subset of enhancers dependent on H3K79 methylation (ATAC-seq)

DOT1L inhibition reveals a distinct subset of enhancers dependent on H3K79 methylation (Capture-C)

DOT1L inhibition reveals a distinct subset of enhancers dependent on H3K79 methylation (ChIP-seq)

DOT1L inhibition reveals a distinct subset of enhancers dependent on H3K79 methylation (RNA-seq)

Determination of an Interaction Network between an Extracellular Bacterial Pathogen and the Human Host

Dynamic chromatin structure during early ILC progenitor differentiation

Exome sequencing identified mutations in clpex mouse mutants

Identification of EOMES-expressing spermatogonial stem cells and their regulation by PLZF

Identification of monocyte-like precursors of granulocytes as a mechanism for accumulation of PMN-MDSC in cancer

Inactivation of Bap1 Cooperates with Losses of Nf2 and Cdkn2a to Drive the Development of Pleural Malignant Mesothelioma in Conditional Mouse Models

Induction of alpha cell-restricted Gc in dedifferentiating beta cells contributes to stress-induced beta-cell dysfunction [ChIP-seq]

Induction of alpha cell-restricted Gc in dedifferentiating beta cells contributes to stress-induced beta-cell dysfunction [RNA-seq]

Inhibition of Phosphoinositide-3-kinase signaling promotes the stem cell state of trophoblast

Lysosomal dysfunction in Down syndrome is APP-dependent and mediated by APP-βCTF (C99)

Quantitative profiling of the UGT transcriptome in human drug metabolizing tissues [Total RNA]

Relative timing of type I interferon response and virus replication determines disease outcome during MERS-CoV infection

Single cell RNA-Seq of sorted nephron progenitor cells from pooled mouse kidneys

Single cell expression analysis reveals anatomical and cell cycle-dependent transcriptional shifts during heart development