JAK-STAT signaling in inflammatory breast cancer enables chemotherapy-resistant cell states [ChIP-seq]

JAK-STAT signaling in inflammatory breast cancer enables chemotherapy-resistant cell states [RNA-seq]

JAK-STAT signaling in inflammatory breast cancer enables chemotherapy-resistant cell states [single-cell RNA-seq]

Breast cancer prevention by short-term inhibition of TGFβ signaling

Rat model of breast cancer to dissect mechanisms of immune escape and immunotherapy sensitivity

Divergent resistance mechanisms to HER2-targeted therapies in breast cancer (bulkRNA-Seq and Exome-Seq)

Divergent resistance mechanisms to HER2-targeted therapies in breast cancer (scRNA-Seq)

Genomic alterations during the in situ to invasive ductal breast carcinoma transition shaped by the immune system

Acquired resistance to combined BET and CDK4/6 inhibition in triple-negative breast cancer [Barcode]

Acquired resistance to combined BET and CDK4/6 inhibition in triple-negative breast cancer [Exome]

Acquired resistance to combined BET and CDK4/6 inhibition in triple-negative breast cancer [RNA-Seq]

Acquired resistance to combined BET and CDK4/6 inhibition in triple-negative breast cancer [scRNA-Seq]

Synthetic lethal and resistance interactions with BET bromodomain inhibitors [ATAC-Seq]

Synthetic lethal and resistance interactions with BET bromodomain inhibitors [Barcode_seq]

Synthetic lethal and resistance interactions with BET bromodomain inhibitors [CRISPR screen]

Synthetic lethal and resistance interactions with BET bromodomain inhibitors [ChIP-Seq]

Synthetic lethal and resistance interactions with BET bromodomain inhibitors [RNA-Seq]

Synthetic lethal and resistance interactions with BET bromodomain inhibitors [scRNA-Seq]

Subclonal cooperation drives metastasis through modulating local and systemic immune microenvironments

The EN1 transcription factor drives neural features and brain metastases in triple negative breats cancer (TNBC)