RNA-seq data of aortas from WT or PALMD+/- mouse

Test case data for ChIC - ChIP-seq quality control pipeline

METTL3-Mediated m6A Modification of TIMP2 mRNA Promotes Podocyte Injury in Diabetic Nephropathy [RNA-seq]

Piezo1 mediated mechanotransduction modulates macrophages proliferation during bone remodeling via PI3K-AKT-Ccnd1 axis

Gene expression changes in in vitro equivalents of DC progenitors from uninfected or LCMV Cl13-infected mice upon Gmeb1 knockdown and treatment with glucocorticoid

Pharmaceutical reversal of Dkk1-induced LRP6 endocytosis prevents cancer cachexia and prolongs survival

Ablation of the Sost gene prevents hallmarks of calcific aortic valve disease in a high-cholesterol mouse model

Cytokine-sensing primary enhancer cluster launches secondary enhancers to activate multi-gene locus [4C-seq]

Cytokine-sensing primary enhancer cluster launches secondary enhancers to activate multi-gene locus [Hi-C]

MRAP mediated adipocyte differentiation by thymic mesenchymal stromal cells contributes to thymic involution

The role of cadherin-11 in Unx/AngII induced kidney injury [Huffstater_AA]

The role of cadherin-11 in Unx/AngII induced kidney injury [Huffstater_Unx]

ChIP-DIP maps binding of hundreds of proteins to DNA simultaneously and identifies diverse gene regulatory elements

RNA sequencing of wild type or calcineurin-deficient mouse bone marrow-derived dendritic cells stimulated with or without lipoarabinomannan (LAM) from Mycobacterium tuberculosis Aoyama-B

CUT&RUN to map WT1 binding in M15 cell lines that are genome edited for the KTS isoforms.

Bulk RNA sequencing and analysis of sorted Common Lymphoid Progenitor (CLP) cells from WT, Notch1+/-, and Notch1+/ΔTAD adult mouse bone marrow

Epithelial deletion of CUL4B increased myeloid-derived suppressor cells-mediated immunosuppression to promote ApcMin/+ tumor formation

Setd2 reprograns lipid metabolism for the resolution of inflammation via H3K36me3 modification

Transcriptome analysis of ear cartilage lipochondrocytes at multiple stages of differentiation.

High-mobility group box1 peptide ameliorates bronchopulmonary dysplasia (BPD) via suppressing inflammation and fibrosis in a mouse model.