PacBio SMRT-seq of wild-type, ∆metJ, and ∆dam Salmonella enterica serovar Typhimurium (14028s)

PacBio SMRT-seq of wild-type, ∆metJ, and ∆yhdJ Salmonella enterica serovar Typhimurium (14028s)

RNA-seq of wild-type and ∆yhdJ Salmonella enterica serovar Typhimurium (14028s)

RNA-seq of wild-type and ∆metJ Salmonella enterica serovar Typhimurium (14028s)

Transcriptional profiling to elucidate processes regulated by 2',3'-cyclic nucleotide phosphodiesterase (CNPase) in Salmonella Typhimurium 14028s (Salmonella) II.

Transcriptional profiling to elucidate processes regulated by 2',3'-cyclic nucleotide phosphodiesterase (CNPase) in Salmonella Typhimurium 14028s (Salmonella) I.

Transcriptional Sequencing of Salmonella enterica serovar Typhimurium wild typle and DsrA-deletion mutant Survival unfer LB or H2O2 conditions

Mammalian apoptotic factors regulate bacterial gene expression II

Mammalian apoptotic factors regulate bacterial gene expression

Transcriptional profiling to elucidate processes regulated by 2',3'-cyclic nucleotide monophosphates in Salmonella enterica serovar Typhimurium (S. Typhimurium)

Genes affected by L-arabinose in Salmonella Typhimurium biofilms

An antibiofilm effect against Salmonella typhimurium rdar biofilm formation upon treatment with the macrolide clarithromycin

Evaluating the effect of spaceflight on the host-pathogen interaction between human intestinal epithelial cells and Salmonella Typhimurium

Global RNA profiles show target selectivity and physiological effects of peptide-delivered antisense antibiotics

Expression data of wild-type Salmonella enterica serovar Typhimurium strain 14028s treated with or without mitomycin C (3 μM)

Transcriptomic Comparison of Common Nontyphoidal Salmonella Serovars Provides Greater Resolution for Understanding Differences in Virulence

RNA-sequencing of dephostatin-treated Salmonella

Spaceflight analogue culture enhances the host-pathogen interaction between Salmonella and a 3-D biomimetic intestinal co-culture model

Host metabolic reprogramming during immune activation promotes intracellular bacterial survival (glycerol or succinate)

Host metabolic reprogramming during immune activation promotes intracellular bacterial survival (2DG)