NCP26 is a potent prolyl-tRNA synthetase inhibitor and induces pro- apoptotic responses with in vivo potency in multiple myeloma. Bulk RNA-seq data.

NCP26 is a potent prolyl-tRNA synthetase inhibitor and induces pro- apoptotic responses with in vivo potency in multiple myeloma. Single-cell RNA-seq data BM samples.

NCP26 is a potent prolyl-tRNA synthetase inhibitor and induces pro- apoptotic responses with in vivo potency in multiple myeloma

Dementia with Lewy bodies post-mortem brains reveal differentially methylated CpG sites with biomarker potential

Single-cell transcriptomics reveals common epithelial response patterns in human acute kidney injury

Genome-wide H3k27ac levels following FOXA1 KO in UMUC-1 human bladder cancer cells [ChIP-seq]

Genome-wide H3k27ac levels following FOXA1 KO in UMUC-1 human bladder cancer cells [RNA-seq]

Genome-wide H3k27ac levels following FOXA1 KO in UMUC-1 human bladder cancer cells

Genome-wide H3k27ac levels following FOXA1 KO in UMUC-1 human bladder cancer cells [scRNA-Seq]

Efficacy-enhanced anti-CD7 universal chimeric antigen receptor-T cell therapy for relapsed/refractory CD7-positive hematological malignancies: A phase I clinical study

Knock-out of 5-lipoxygenase in overexpressing tumor cells – consequences on gene expression and cellular function

RPRD1B is required for proper heat shock response [TTchem-seq]

RPRD1B is required for proper heat shock response [RNA-seq]

RPRD1B is required for proper heat shock response

Single-cell RNA sequencing of MCF-7, T-47D and ZR-75-1 breast cancer cell lines treated with estrogen and progesterone

Gene expression profile of primary CLL cells transduced with a retrovirus expressing mutated NOTCH1

Gene expression profile of primary CLL cells transduced with a mutated form on NOTCH1 following IgM stimulation

Gene expression profile of primary CLL cells transduced with a mutated NOTCH1

Histone H3 lysine 27 acetylation (H3K27ac) profile of primary CLL cells transduced with a retrovirus expressing mutated NOTCH1

Stem Cell transcriptional response to Microglia-Conditioned Media