NCBI Logo
GEO Logo
   NCBI > GEO > Accession DisplayHelp Not logged in | LoginHelp
GEO help: Mouse over screen elements for information.
          Go
Series GSE108332 Query DataSets for GSE108332
Status Public on Apr 02, 2019
Title Next Generation Sequencing Facilitates Quantitative Analysis of mRNA expression of Wild Type and Mettl3-/- mice
Organism Mus musculus
Experiment type Expression profiling by high throughput sequencing
Summary Considering m6A modification in mRNA was reported to be important in many biology processes, like cell fate decision and embryonic development and was closely connected to human diseases such as metabolic diseases, neuron disorders and cancer. We wondered how Mettl3 affect dendritic cell development.
 
Overall design Replicate representing WT and KO dendritic cells stimulated with LPS were analyzed
 
Contributor(s) Cao X, Wang H, Hu X
Citation(s) 31015515
Submission date Dec 20, 2017
Last update date May 01, 2019
Contact name Huamin Wang
E-mail(s) huamin_wang86@163.com
Phone +8618017766823
Organization name Peking Union Medical College, Chinese Academy of Medical Sciences,
Department Department of Immunology
Street address Three N0. 5 dongdan street, Dongcheng District, Beijing
City Beijing, China
ZIP/Postal code 100730
Country China
 
Platforms (1)
GPL21493 Illumina HiSeq 3000 (Mus musculus)
Samples (4)
GSM2895440 KO-RNA seq-rep1
GSM2895441 WT-RNA seq-rep1
GSM2895442 KO-RNA seq-rep2
This SubSeries is part of SuperSeries:
GSE108333 Mettl3-mediated mRNA m6A methylation promotes dendritic cell activation
Relations
BioProject PRJNA423187
SRA SRP127228

Download family Format
SOFT formatted family file(s) SOFTHelp
MINiML formatted family file(s) MINiMLHelp
Series Matrix File(s) TXTHelp

Supplementary file Size Download File type/resource
GSE108332_RNA_seq-rep1.xls.gz 2.0 Mb (ftp)(http) XLS
GSE108332_RNA_seq-rep2.xls.gz 1021.2 Kb (ftp)(http) XLS
SRA Run SelectorHelp
Raw data are available in SRA
Processed data are available on Series record

| NLM | NIH | GEO Help | Disclaimer | Accessibility |
NCBI Home NCBI Search NCBI SiteMap