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Status |
Public on Jun 22, 2022 |
Title |
SME1 deficiency results in oxidative stress tolerance |
Organism |
Arabidopsis thaliana |
Experiment type |
Expression profiling by high throughput sequencing
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Summary |
In this work, we describe a nonsense mutation in SME1 that alleviates photorespiratory cell death in the Arabidopsis thaliana cat2-2 mutant. A SME1 loss-of-function mutant (sme1-1) and wild-type Col-0 were subjected to mRNA-seq for differential gene expression and alternative splicing analysis. This revealed a massive transcript reprogramming and occurrence of splicing defects caused by SME1 deficiency. For more details see the publication.
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Overall design |
2 genotypes: sme1-1 and wild-type Col-0 X 3 replicates = 6 samples
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Contributor(s) |
Van der Kelen K, Van Ruyskensvelde V, Willems P, Van Breusegem F |
Citation missing |
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Submission date |
Jun 22, 2019 |
Last update date |
Jun 22, 2022 |
Contact name |
Patrick Willems |
E-mail(s) |
pawil@psb.ugent.be
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Organization name |
Ghent University
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Department |
PSB
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Street address |
Technologiepark 927
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City |
Gent |
ZIP/Postal code |
9052 |
Country |
Belgium |
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Platforms (1) |
GPL19580 |
Illumina NextSeq 500 (Arabidopsis thaliana) |
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Samples (6)
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Relations |
BioProject |
PRJNA550206 |
SRA |
SRP211749 |