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Series GSE196865 Query DataSets for GSE196865
Status Public on Mar 31, 2022
Title Macrophage-produced VEGFC is induced by efferocytosis to resolve inflammation and improve ventricular function after myocardial infarction (ChIP-Seq)
Organism Mus musculus
Experiment type Genome binding/occupancy profiling by high throughput sequencing
Summary Recent reports have suggested a protective role for vascular endothelial growth factor C (VEGFC) during acute cardiac lymphangiogenesis post MI. Glinton et al. report that defective efferocytosis by macrophages after experimental MI leads to a reduction in cardiac lymphangiogenesis and Vegfc expression.
 
Overall design H3K27Ac ChIP-sequencing of C57BL6J bone marrow derived macrophages after incubation with UV-treated apoptotic Jurkat cells for 6 hours
Web link https://pubmed.ncbi.nlm.nih.gov/35271504/
 
Contributor(s) Glinton K, Lantz C, Thorp EB
Citation(s) 35271504
Submission date Feb 16, 2022
Last update date Jun 30, 2022
Contact name Connor Lantz
E-mail(s) connor.lantz@northwestern.edu
Organization name Northwestern University
Street address 303 E. Chicago Ave. Ward 4-075
City Chicago
State/province IL
ZIP/Postal code 60611
Country USA
 
Platforms (1)
GPL19057 Illumina NextSeq 500 (Mus musculus)
Samples (2)
GSM5903326 Control
GSM5903327 ACs
This SubSeries is part of SuperSeries:
GSE196867 Macrophage-produced VEGFC is induced by efferocytosis to resolve inflammation and improve ventricular function after myocardial infarction
Relations
BioProject PRJNA807634

Download family Format
SOFT formatted family file(s) SOFTHelp
MINiML formatted family file(s) MINiMLHelp
Series Matrix File(s) TXTHelp

Supplementary file Size Download File type/resource
GSE196865_ChipSeq_Output.xlsx 8.8 Mb (ftp)(http) XLSX
SRA Run SelectorHelp
Raw data are available in SRA
Processed data are available on Series record

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