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Status |
Public on Sep 01, 2015 |
Title |
Genome wide analysis of androgen-receptor binding sites and epigenetic condition in prostate cancer |
Organism |
Homo sapiens |
Experiment type |
Genome binding/occupancy profiling by high throughput sequencing
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Summary |
Prostate cancer is the most common cancer in men and AR downstream signalings promote prostate cancer cell proliferation. To investigate the AR signaling, we performed ChIP sequence analysis in AR positive prostate cancer cell line, LNCaP. In addition, we used hormone-refractory prostate cancer model cells, Bicalutamide-resistant (BicR) to explore the differences of androgen signaling in prostate cancer progression.
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Overall design |
ChIP sequence analysis of AR binding sites and epigenetic condition in two prostate cancer cells
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Contributor(s) |
Takayama K, Inoue S |
Citation(s) |
26404510 |
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Submission date |
Feb 18, 2015 |
Last update date |
May 15, 2019 |
Contact name |
Ken-ichi Takayama |
Organization name |
Tokyo Metropolitan Institute of Gerontology
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Street address |
Sakaecho
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City |
Itabashi-ku |
ZIP/Postal code |
173-0015 |
Country |
Japan |
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Platforms (2) |
GPL10999 |
Illumina Genome Analyzer IIx (Homo sapiens) |
GPL11154 |
Illumina HiSeq 2000 (Homo sapiens) |
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Samples (21)
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This SubSeries is part of SuperSeries: |
GSE66039 |
Global analysis of androgen-signaling reveals the function of miRNAs for the epigenomic regulation in prostate cancer cells |
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Relations |
BioProject |
PRJNA275746 |
SRA |
SRP055133 |