Homo sapiens cDNA, FLJ93275, Homo sapiens potassium inwardly-rectifyin - Nucleotide

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The W101C KCNJ5 Mutation Induces Slower Pacing by Constitutively Active GIRK Channels in hiPSC-Derived Cardiomyocytes. [Int J Mol Sci. 2023]
The W101C KCNJ5 Mutation Induces Slower Pacing by Constitutively Active GIRK Channels in hiPSC-Derived Cardiomyocytes.
Kayser A, Dittmann S, Šarić T, Mearini G, Verkerk AO, Schulze-Bahr E. Int J Mol Sci. 2023 Oct 18; 24(20). Epub 2023 Oct 18.
KCNJ5 mutation is a predictor for recovery of endothelial function after adrenalectomy in patients with aldosterone-producing adenoma. [Hypertens Res. 2023]
KCNJ5 mutation is a predictor for recovery of endothelial function after adrenalectomy in patients with aldosterone-producing adenoma.
Kishimoto S, Oki K, Maruhashi T, Kajikawa M, Mizobuchi A, Harada T, Yamaji T, Hashimoto Y, Yoshimura K, Nakano Y, et al. Hypertens Res. 2023 Sep; 46(9):2213-2227. Epub 2023 Jul 18.
Cardiovascular and metabolic characters of KCNJ5 somatic mutations in primary aldosteronism. [Front Endocrinol (Lausanne). 2...]
Cardiovascular and metabolic characters of KCNJ5 somatic mutations in primary aldosteronism.
Chang YY, Lee BC, Chen ZW, Tsai CH, Chang CC, Liao CW, Pan CT, Peng KY, Chou CH, Lu CC, et al. Front Endocrinol (Lausanne). 2023; 14:1061704. Epub 2023 Mar 6.

Homo sapiens cDNA, FLJ93275, Homo sapiens potassium inwardly-rectifying channel,...
Homo sapiens cDNA, FLJ93275, Homo sapiens potassium inwardly-rectifying channel, subfamily J,member 5 (KCNJ5), mRNA
gi|164690639|dbj|AK312837.1|

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Nucleotide