U.S. flag

An official website of the United States government

Display Settings:

Format
Sort by

Send to:

Choose Destination

Links from Nucleotide

Items: 5

1.
GTP hydrolysis promotes disassembly of the atlastin crossover dimer during ER fusion
James Winsor, Ursula Machi, Qixiu Han, David D. Hackney, Tina H. Lee
J Cell Biol. 2018 Dec 3; 217(12): 4184–4198. doi: 10.1083/jcb.201805039
PMCID:
PMC6279388
AbstractArticlePDF–1.9M
2.
The crossover conformational shift of the GTPase atlastin provides the energy driving ER fusion
James Winsor, David D. Hackney, Tina H. Lee
J Cell Biol. 2017 May 1; 216(5): 1321–1335. doi: 10.1083/jcb.201609071
PMCID:
PMC5412568
AbstractArticlePDF–2.2M
3.
Autosomal dominant familial spastic paraplegia: reduction of the FSP1 candidate region on chromosome 14q to 7 cM and locus heterogeneity.
S Gispert, N Santos, R Damen, T Voit, J Schulz, T Klockgether, G Orozco, F Kreuz, J Weissenbach, G Auburger
Am J Hum Genet. 1995 Jan; 56(1): 183–187.
PMCID:
PMC1801298
SummaryPage BrowsePDF–773K
4.
Mutational Spectrum of Spast (Spg4) and Atl1 (Spg3a) Genes In Russian Patients With Hereditary Spastic Paraplegia
V. A. Kadnikova, G. E. Rudenskaya, A. A. Stepanova, I. G. Sermyagina, O. P. Ryzhkova
Sci Rep. 2019; 9: 14412. Published online 2019 Oct 8. doi: 10.1038/s41598-019-50911-9
PMCID:
PMC6783457
AbstractArticlePDF–1.6M
5.
Evidence for autosomal recessive inheritance in SPG3A caused by homozygosity for a novel ATL1 missense mutation
Tahir Naeem Khan, Joakim Klar, Muhammad Tariq, Shehla Anjum Baig, Naveed Altaf Malik, Raja Yousaf, Shahid Mahmood Baig, Niklas Dahl
Eur J Hum Genet. 2014 Oct; 22(10): 1180–1184. Published online 2014 Jan 29. doi: 10.1038/ejhg.2014.5
PMCID:
PMC4169543
AbstractArticlePDF–696K

Display Settings:

Format
Sort by

Send to:

Choose Destination

Supplemental Content

Filter your results:

Find related data

Recent activity

Clear Turn Off Turn On

Your browsing activity is empty.

Activity recording is turned off.

Turn recording back on

See more...
You are here: NCBI > Literature > PubMed Central (PMC)
Support Center