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Coassembly of K(V)LQT1 and minK (IsK) proteins to form cardiac I(Ks) potassium channel.
Nature. 1996 Nov 7;384(6604):80-3. doi: 10.1038/384080a0.
Nature. 1996.
PMID: 8900283
Mutations in the hminK gene cause long QT syndrome and suppress IKs function.
Splawski I, Tristani-Firouzi M, Lehmann MH, Sanguinetti MC, Keating MT.
Splawski I, et al.
Nat Genet. 1997 Nov;17(3):338-40. doi: 10.1038/ng1197-338.
Nat Genet. 1997.
PMID: 9354802
Item in Clipboard
Cellular dysfunction of LQT5-minK mutants: abnormalities of IKs, IKr and trafficking in long QT syndrome.
Bianchi L, Shen Z, Dennis AT, Priori SG, Napolitano C, Ronchetti E, Bryskin R, Schwartz PJ, Brown AM.
Bianchi L, et al.
Hum Mol Genet. 1999 Aug;8(8):1499-507. doi: 10.1093/hmg/8.8.1499.
Hum Mol Genet. 1999.
PMID: 10400998
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AKAP proteins anchor cAMP-dependent protein kinase to KvLQT1/IsK channel complex.
Potet F, Scott JD, Mohammad-Panah R, Escande D, Baró I.
Potet F, et al.
Am J Physiol Heart Circ Physiol. 2001 May;280(5):H2038-45. doi: 10.1152/ajpheart.2001.280.5.H2038.
Am J Physiol Heart Circ Physiol. 2001.
PMID: 11299204
Free article.
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KCNQ1 gain-of-function mutation in familial atrial fibrillation.
Chen YH, Xu SJ, Bendahhou S, Wang XL, Wang Y, Xu WY, Jin HW, Sun H, Su XY, Zhuang QN, Yang YQ, Li YB, Liu Y, Xu HJ, Li XF, Ma N, Mou CP, Chen Z, Barhanin J, Huang W.
Chen YH, et al.
Science. 2003 Jan 10;299(5604):251-4. doi: 10.1126/science.1077771.
Science. 2003.
PMID: 12522251
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Augmented potassium current is a shared phenotype for two genetic defects associated with familial atrial fibrillation.
Abraham RL, Yang T, Blair M, Roden DM, Darbar D.
Abraham RL, et al.
J Mol Cell Cardiol. 2010 Jan;48(1):181-90. doi: 10.1016/j.yjmcc.2009.07.020. Epub 2009 Jul 30.
J Mol Cell Cardiol. 2010.
PMID: 19646991
Free PMC article.
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