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1975
2025

Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1975 1
1977 1
1979 1
1988 4
1990 3
1991 2
1992 5
1993 6
1994 7
1996 10
1997 6
1998 6
1999 7
2000 7
2001 10
2002 13
2003 4
2004 11
2005 5
2006 10
2007 11
2008 15
2009 11
2010 16
2011 9
2012 17
2013 22
2014 12
2015 18
2016 13
2017 24
2018 28
2019 18
2020 15
2021 20
2022 6
2023 13
2024 15
2025 0

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357 results

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Page 1
Astrocytes: biology and pathology.
Sofroniew MV, Vinters HV. Sofroniew MV, et al. Acta Neuropathol. 2010 Jan;119(1):7-35. doi: 10.1007/s00401-009-0619-8. Epub 2009 Dec 10. Acta Neuropathol. 2010. PMID: 20012068 Free PMC article. Review.
Substantial progress has been made recently in determining functions and mechanisms of reactive astrogliosis and in identifying roles of astrocytes in CNS disorders and pathologies. A vast molecular arsenal at the disposal of reactive astrocytes is being defined. .. …
Substantial progress has been made recently in determining functions and mechanisms of reactive astrogliosis and in identifying roles of …
Microglia in depression: current perspectives.
Jia X, Gao Z, Hu H. Jia X, et al. Sci China Life Sci. 2021 Jun;64(6):911-925. doi: 10.1007/s11427-020-1815-6. Epub 2020 Oct 14. Sci China Life Sci. 2021. PMID: 33068286 Review.
Microglia respond to stress-triggered neuroinflammation, and through the release of proinflammatory cytokines and their metabolic products, microglia may modulate the function of neurons and astrocytes to regulate depression. In this review, we focused on the role of micro …
Microglia respond to stress-triggered neuroinflammation, and through the release of proinflammatory cytokines and their metabolic products, …
Neuron-Glia Signaling in Synapse Elimination.
Wilton DK, Dissing-Olesen L, Stevens B. Wilton DK, et al. Annu Rev Neurosci. 2019 Jul 8;42:107-127. doi: 10.1146/annurev-neuro-070918-050306. Annu Rev Neurosci. 2019. PMID: 31283900 Review.
Without proper functioning of these cells, the number, morphology, and function of synaptic contacts are profoundly altered, resulting in abnormal connectivity and behavioral abnormalities. ...
Without proper functioning of these cells, the number, morphology, and function of synaptic contacts are profoundly altered, resultin …
Astrocytic and microglial cells as the modulators of neuroinflammation in Alzheimer's disease.
Singh D. Singh D. J Neuroinflammation. 2022 Aug 17;19(1):206. doi: 10.1186/s12974-022-02565-0. J Neuroinflammation. 2022. PMID: 35978311 Free PMC article. Review.
Studies have shown that microglia and astrocytes undergo alterations in their genetic profile, cellular and molecular responses, and thus promote dysfunctional immune cross-talk in AD. Hence, targeting microglia and astrocytes-driven molecular pathways could resolve …
Studies have shown that microglia and astrocytes undergo alterations in their genetic profile, cellular and molecular responses, and …
Myelination of the nervous system: mechanisms and functions.
Nave KA, Werner HB. Nave KA, et al. Annu Rev Cell Dev Biol. 2014;30:503-33. doi: 10.1146/annurev-cellbio-100913-013101. Annu Rev Cell Dev Biol. 2014. PMID: 25288117 Review.
In the central nervous system, myelination is also stimulated by axonal activity and astrocytes, whereas myelin clearance involves microglia/macrophages. Once myelinated, the long-term integrity of axons depends on glial supply of metabolites and neurotrophic factors. ...
In the central nervous system, myelination is also stimulated by axonal activity and astrocytes, whereas myelin clearance involves mi …
Functional morphology of the blood-brain barrier in health and disease.
Liebner S, Dijkhuizen RM, Reiss Y, Plate KH, Agalliu D, Constantin G. Liebner S, et al. Acta Neuropathol. 2018 Mar;135(3):311-336. doi: 10.1007/s00401-018-1815-1. Epub 2018 Feb 6. Acta Neuropathol. 2018. PMID: 29411111 Free PMC article. Review.
The adult quiescent blood-brain barrier (BBB), a structure organised by endothelial cells through interactions with pericytes, astrocytes, neurons and microglia in the neurovascular unit, is highly regulated but fragile at the same time. ...Throughout the review, we highli …
The adult quiescent blood-brain barrier (BBB), a structure organised by endothelial cells through interactions with pericytes, astrocytes
Glioblastoma: pathology, molecular mechanisms and markers.
Aldape K, Zadeh G, Mansouri S, Reifenberger G, von Deimling A. Aldape K, et al. Acta Neuropathol. 2015 Jun;129(6):829-48. doi: 10.1007/s00401-015-1432-1. Epub 2015 May 6. Acta Neuropathol. 2015. PMID: 25943888 Review.
The current WHO definition of GBM relies on the presence of high-grade astrocytic neoplasm with the presence of either microvascular proliferation and/or tumor necrosis. ...In this review, we describe major clinically relevant genetic and epigenetic abnormalities in …
The current WHO definition of GBM relies on the presence of high-grade astrocytic neoplasm with the presence of either microvascular …
Molecular pathophysiology of cerebral edema.
Stokum JA, Gerzanich V, Simard JM. Stokum JA, et al. J Cereb Blood Flow Metab. 2016 Mar;36(3):513-38. doi: 10.1177/0271678X15617172. Epub 2015 Nov 16. J Cereb Blood Flow Metab. 2016. PMID: 26661240 Free PMC article. Review.
Following acute CNS injury, cells of the neurovascular unit, particularly brain endothelial cells and astrocytes, undergo a program of pre- and post-transcriptional changes in the activity of ion channels and transporters. These changes can result in maladaptive ion transp …
Following acute CNS injury, cells of the neurovascular unit, particularly brain endothelial cells and astrocytes, undergo a program o …
Glial cells in schizophrenia: a unified hypothesis.
Dietz AG, Goldman SA, Nedergaard M. Dietz AG, et al. Lancet Psychiatry. 2020 Mar;7(3):272-281. doi: 10.1016/S2215-0366(19)30302-5. Epub 2019 Nov 6. Lancet Psychiatry. 2020. PMID: 31704113 Free PMC article. Review.
We discuss neuroimaging studies, pathological findings, and experimental work supporting the idea that glial cells might contribute to the development of schizophrenia. Experimental studies suggest that abnormalities in the differentiation competence of glial progenitor ce …
We discuss neuroimaging studies, pathological findings, and experimental work supporting the idea that glial cells might contribute to the d …
The neuropathology of chronic traumatic encephalopathy.
McKee AC, Stein TD, Kiernan PT, Alvarez VE. McKee AC, et al. Brain Pathol. 2015 May;25(3):350-64. doi: 10.1111/bpa.12248. Brain Pathol. 2015. PMID: 25904048 Free PMC article. Review.
CTE is a tauopathy characterized by the deposition of hyperphosphorylated tau (p-tau) protein as neurofibrillary tangles, astrocytic tangles and neurites in striking clusters around small blood vessels of the cortex, typically at the sulcal depths. Severely affected cases …
CTE is a tauopathy characterized by the deposition of hyperphosphorylated tau (p-tau) protein as neurofibrillary tangles, astrocytic
357 results