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1978
2025

Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1978 1
1986 1
1988 2
1992 1
1993 4
1994 1
1995 2
1998 2
2000 2
2001 2
2002 3
2003 3
2004 3
2005 2
2006 4
2007 1
2008 2
2009 1
2010 1
2011 3
2012 3
2013 2
2014 7
2015 4
2016 6
2017 5
2018 1
2019 4
2020 5
2021 5
2022 1
2023 4
2024 1
2025 0

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80 results

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Page 1
White Matter Diseases with Radiologic-Pathologic Correlation.
Sarbu N, Shih RY, Jones RV, Horkayne-Szakaly I, Oleaga L, Smirniotopoulos JG. Sarbu N, et al. Radiographics. 2016 Sep-Oct;36(5):1426-47. doi: 10.1148/rg.2016160031. Radiographics. 2016. Update in: Radiographics. 2020 May-Jun;40(3):E4-E7. doi: 10.1148/rg.2020190204 PMID: 27618323 Updated. Review.
White matter diseases include a wide spectrum of disorders that have in common impairment of normal myelination, either by secondary destruction of previously myelinated structures (demyelinating processes) or by primary abnormalities of myelin formation (dysmyelinating pr …
White matter diseases include a wide spectrum of disorders that have in common impairment of normal myelination, either by secondary destruc …
An update on clinical, pathological, diagnostic, and therapeutic perspectives of childhood leukodystrophies.
Ashrafi MR, Amanat M, Garshasbi M, Kameli R, Nilipour Y, Heidari M, Rezaei Z, Tavasoli AR. Ashrafi MR, et al. Expert Rev Neurother. 2020 Jan;20(1):65-84. doi: 10.1080/14737175.2020.1699060. Epub 2019 Dec 12. Expert Rev Neurother. 2020. PMID: 31829048 Review.
Comprehensive evaluation of clinical findings, brain magnetic resonance imaging, and genetic studies play the key roles in the early diagnosis of individuals with leukodystrophies. ...New genetic methods and stem cell transplantation are also under investigation to further …
Comprehensive evaluation of clinical findings, brain magnetic resonance imaging, and genetic studies play the key roles in the early …
Biology of oligodendrocyte and myelin in the mammalian central nervous system.
Baumann N, Pham-Dinh D. Baumann N, et al. Physiol Rev. 2001 Apr;81(2):871-927. doi: 10.1152/physrev.2001.81.2.871. Physiol Rev. 2001. PMID: 11274346 Free article. Review.
The best examples are the studies concerning the effects of the mutations affecting the most abundant protein in the central nervous myelin, the proteolipid protein, which lead to dysmyelinating diseases in animals and human (jimpy mutation and Pelizaeus-Merzbacher disease …
The best examples are the studies concerning the effects of the mutations affecting the most abundant protein in the central nervous myelin, …
Is psychosis a dysmyelination-related information-processing disorder?
Giotakos O. Giotakos O. Psychiatriki. 2019 Jul-Sep;30(3):245-255. doi: 10.22365/jpsych.2019.303.245. Psychiatriki. 2019. PMID: 31685456 Free article. Review.
A growing body of evidence points towards the involvement of dysmyelination of the prefrontal cortex in the development of the cognitive symptoms of psychosis. ...The dysmyelination-induced delays in patients with psychosis may cause a discrepancy in sensory feedbac …
A growing body of evidence points towards the involvement of dysmyelination of the prefrontal cortex in the development of the cognit …
Microglia-Mediated Neurodegeneration in Perinatal Brain Injuries.
Fleiss B, Van Steenwinckel J, Bokobza C, K Shearer I, Ross-Munro E, Gressens P. Fleiss B, et al. Biomolecules. 2021 Jan 13;11(1):99. doi: 10.3390/biom11010099. Biomolecules. 2021. PMID: 33451166 Free PMC article. Review.
The nature and severity of changes vary according to the type and severity of insult and maturation stage of the brain. Microglial activation has been demonstrated almost ubiquitously in perinatal brain injuries and these responses are key cell orchestrators of b
The nature and severity of changes vary according to the type and severity of insult and maturation stage of the brain. Microglial ac …
Oligodendrocyte pathology in Huntington's disease: from mechanisms to therapeutics.
Ferrari Bardile C, Radulescu CI, Pouladi MA. Ferrari Bardile C, et al. Trends Mol Med. 2023 Oct;29(10):802-816. doi: 10.1016/j.molmed.2023.07.010. Epub 2023 Aug 15. Trends Mol Med. 2023. PMID: 37591764 Review.
White matter (WM) structures, representing myelin-rich regions of the brain, are profoundly affected in HD, and recent findings reveal oligodendroglia dysfunction as an early pathological event. Here, we focus on mechanisms that underlie oligodendroglial deficits and dy
White matter (WM) structures, representing myelin-rich regions of the brain, are profoundly affected in HD, and recent findings revea …
Dysmyelination revisited.
Poser CM. Poser CM. Arch Neurol. 1978 Jul;35(7):401-8. doi: 10.1001/archneur.1978.00500310003001. Arch Neurol. 1978. PMID: 352308 Review.
Dysmyelination describes an inborn error of metabolism affecting myelinogenesis that causes it to be abnormal, arrested, or delayed. Abiotrophy or myelin as defined by Gowers, due to metabolic failure of the myelin maintenance system, is yet another feature of dysmyelin
Dysmyelination describes an inborn error of metabolism affecting myelinogenesis that causes it to be abnormal, arrested, or delayed.
Severe Convulsions and Dysmyelination in Both Jimpy and Cx32/47 (-/-) Mice may Associate Astrocytic L-Channel Function with Myelination and Oligodendrocytic Connexins with Internodal K(v) Channels.
Chaban YHG, Chen Y, Hertz E, Hertz L. Chaban YHG, et al. Neurochem Res. 2017 Jun;42(6):1747-1766. doi: 10.1007/s11064-017-2194-z. Epub 2017 Feb 18. Neurochem Res. 2017. PMID: 28214987 Review.
A normally occurring increase in oxygen consumption by highly elevated K(+) concentrations is absent in Jimpy brain slices and cultured astrocytes, reflecting that Plp at early embryonic stages affects common precursors as also shown by the ability of conditioned medium fr …
A normally occurring increase in oxygen consumption by highly elevated K(+) concentrations is absent in Jimpy brain slices and cultur …
Myelin in Alzheimer's disease: culprit or bystander?
Maitre M, Jeltsch-David H, Okechukwu NG, Klein C, Patte-Mensah C, Mensah-Nyagan AG. Maitre M, et al. Acta Neuropathol Commun. 2023 Mar 31;11(1):56. doi: 10.1186/s40478-023-01554-5. Acta Neuropathol Commun. 2023. PMID: 37004127 Free PMC article. Review.
While familial AD is caused by specific mutations, the sporadic disease is more common and appears to result from a complex chronic brain neuroinflammation with mitochondriopathies, inducing free radicals' accumulation. ...We propose that some pathophysiological and clinic …
While familial AD is caused by specific mutations, the sporadic disease is more common and appears to result from a complex chronic brain
RNA metabolism and dysmyelination in schizophrenia.
McInnes LA, Lauriat TL. McInnes LA, et al. Neurosci Biobehav Rev. 2006;30(4):551-61. doi: 10.1016/j.neubiorev.2005.10.003. Epub 2006 Jan 30. Neurosci Biobehav Rev. 2006. PMID: 16445981 Review.
Decreased expression of a subset of oligodendrocyte and myelin-related genes is the most consistent finding among gene expression studies of postmortem brain tissue from subjects with schizophrenia (SCZ), although heritable variants have yet to be found that can explain th …
Decreased expression of a subset of oligodendrocyte and myelin-related genes is the most consistent finding among gene expression studies of …
80 results