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Year Number of Results
1990 1
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2008 1
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Page 1
STRIPAK complexes: structure, biological function, and involvement in human diseases.
Hwang J, Pallas DC. Hwang J, et al. Int J Biochem Cell Biol. 2014 Feb;47:118-48. doi: 10.1016/j.biocel.2013.11.021. Epub 2013 Dec 11. Int J Biochem Cell Biol. 2014. PMID: 24333164 Free PMC article. Review.
Moreover, they appear to be regulatory/targeting subunits for the major eukaryotic serine/threonine protein phosphatase 2A. In addition, striatin family members associate with germinal center kinase III kinases as well as other novel components, earning these assemblies th …
Moreover, they appear to be regulatory/targeting subunits for the major eukaryotic serine/threonine protein phosphatase 2A. In additi …
Unsolved medical issues and new targets for further research in viral myocarditis and dilated cardiomyopathy.
Knowlton KU. Knowlton KU. Ernst Schering Res Found Workshop. 2006;(55):19-35. doi: 10.1007/3-540-30822-9_2. Ernst Schering Res Found Workshop. 2006. PMID: 16329655 Review.
Meaningful advances have been made in understanding the mechanisms that contribute to dilated cardiomyopathy and myocarditis. Our data confirmed the hypothesis that there is an interaction of genetic predisposition and acquired factors, in that both can affect the d …
Meaningful advances have been made in understanding the mechanisms that contribute to dilated cardiomyopathy and myocarditis. …
CVB infection and mechanisms of viral cardiomyopathy.
Knowlton KU. Knowlton KU. Curr Top Microbiol Immunol. 2008;323:315-35. doi: 10.1007/978-3-540-75546-3_15. Curr Top Microbiol Immunol. 2008. PMID: 18357777 Review.
Coxsackievirus infection has been demonstrated to be a cause of acute and fulminant viral myocarditis and has been associated with dilated cardiomyopathy. While considerable attention has focused on the role of the cellular and humoral, antigen-specific immune syste …
Coxsackievirus infection has been demonstrated to be a cause of acute and fulminant viral myocarditis and has been associated with dilate
Enteroviral cardiomyopathy: bad news for the dystrophin-glycoprotein complex.
Badorff C, Lee GH, Knowlton KU. Badorff C, et al. Herz. 2000 May;25(3):227-32. doi: 10.1007/s000590050011. Herz. 2000. PMID: 10904843 Review.
Genetic deficiency of the dystrophin-glycoprotein complex causes hereditary dilated cardiomyopathy. Enteroviruses can also cause cardiomyopathy and we have recently described a potential molecular mechanism for enterovirus-induced dilated cardiomyop
Genetic deficiency of the dystrophin-glycoprotein complex causes hereditary dilated cardiomyopathy. Enteroviruses can also cau …
Conditioned nutritional requirements: therapeutic relevance to heart failure.
Sole MJ, Jeejeebhoy KN. Sole MJ, et al. Herz. 2002 Mar;27(2):174-8. doi: 10.1007/s00059-002-2360-0. Herz. 2002. PMID: 12025462 Review.
Deficiencies of carnitine or taurine alone are well documented to result in dilated cardiomyopathy in animals and humans. Each of these deficiencies is amenable to restoration through dietary supplementation. ...METHOD AND RESULTS: We have demonstrated deficiencies …
Deficiencies of carnitine or taurine alone are well documented to result in dilated cardiomyopathy in animals and humans. Each …
Fatal cardiomyopathy associated with pegylated interferon/ribavirin in a patient with chronic hepatitis C.
Condat B, Asselah T, Zanditenas D, Estampes B, Cohen A, O'Toole D, Bonnet J, Ngo Y, Marcellin P, Blazquez M. Condat B, et al. Eur J Gastroenterol Hepatol. 2006 Mar;18(3):287-9. doi: 10.1097/00042737-200603000-00010. Eur J Gastroenterol Hepatol. 2006. PMID: 16462543 Review.
The relationship between pegylated interferon-alpha/ribavirin and the development of cardiomyopathy is highly probable for the following reasons: (1) a cardiologist consultation with specific investigations had been performed before treatment excluding a pre-existing ca
The relationship between pegylated interferon-alpha/ribavirin and the development of cardiomyopathy is highly probable for the follow …
Rigid spine syndrome associated with cardiomyopathy: clinical and nosological considerations.
Takase Y, Yamamoto K, Nogaki H, Fukusako T, Sasabe F, Morimatsu M. Takase Y, et al. Jpn J Med. 1990 Sep-Oct;29(5):555-9. doi: 10.2169/internalmedicine1962.29.555. Jpn J Med. 1990. PMID: 2089184 Free article. Review.
We report observations in a 32-year-old man with the following characteristics of rigid spine syndrome: humero-peroneal muscular atrophy and weakness; bradycardia, dilated cardiomegaly and complete cardiac conduction block; and severe fatty degeneration of the paravertebra …
We report observations in a 32-year-old man with the following characteristics of rigid spine syndrome: humero-peroneal muscular atrophy and …