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1986 1
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Page 1
Side effects of long-term glutamine supplementation.
Holecek M. Holecek M. JPEN J Parenter Enteral Nutr. 2013 Sep;37(5):607-16. doi: 10.1177/0148607112460682. Epub 2012 Sep 18. JPEN J Parenter Enteral Nutr. 2013. PMID: 22990615 Review.
The following side effects of GLN supplementation are discussed: (1) Alterations in amino acid transport-as GLN shares the transporters with other amino acids, enhanced GLN intake may impair amino acid distribution among tissues and their absorption in the gut and k …
The following side effects of GLN supplementation are discussed: (1) Alterations in amino acid transport-as GLN shares the transpo …
Inherited copper transport disorders: biochemical mechanisms, diagnosis, and treatment.
Kodama H, Fujisawa C, Bhadhprasit W. Kodama H, et al. Curr Drug Metab. 2012 Mar;13(3):237-50. doi: 10.2174/138920012799320455. Curr Drug Metab. 2012. PMID: 21838703 Free PMC article. Review.
Meanwhile, connective tissue disorders cannot be improved by copper-histidine treatment. Combination therapy with copper-histidine injections and oral administration of disulfiram is being investigated. Occipital horn syndrome characterized by connective tissue a …
Meanwhile, connective tissue disorders cannot be improved by copper-histidine treatment. Combination therapy with copper-histidine …
The folate cycle and disease in humans.
Fowler B. Fowler B. Kidney Int Suppl. 2001 Feb;78:S221-9. doi: 10.1046/j.1523-1755.2001.59780221.x. Kidney Int Suppl. 2001. PMID: 11169015 Free article. Review.
Vital processes in folate disposition include conversion of dietary folylpolyglutamates to monoglutamates, intestinal absorption, receptor and carrier-mediated transport across cell membranes, and cellular export. Folate coenzymes are responsible for the one-carbon unit tr …
Vital processes in folate disposition include conversion of dietary folylpolyglutamates to monoglutamates, intestinal absorption, receptor a …
Chelating principles in Menkes and Wilson diseases: Choosing the right compounds in the right combinations at the right time.
Horn N, Møller LB, Nurchi VM, Aaseth J. Horn N, et al. J Inorg Biochem. 2019 Jan;190:98-112. doi: 10.1016/j.jinorgbio.2018.10.009. Epub 2018 Oct 23. J Inorg Biochem. 2019. PMID: 30384011 Review.
Dysregulation of copper homeostasis in humans is primarily found in two genetic diseases of copper transport, Menkes and Wilson diseases, which show symptoms of copper deficiency or overload, respectively. ...Therapy of Menkes disease with copper-histidine, thiocarb …
Dysregulation of copper homeostasis in humans is primarily found in two genetic diseases of copper transport, Menkes and Wilson disea …
Ascending monoaminergic systems alterations in Alzheimer's disease. translating basic science into clinical care.
Trillo L, Das D, Hsieh W, Medina B, Moghadam S, Lin B, Dang V, Sanchez MM, De Miguel Z, Ashford JW, Salehi A. Trillo L, et al. Neurosci Biobehav Rev. 2013 Sep;37(8):1363-79. doi: 10.1016/j.neubiorev.2013.05.008. Epub 2013 May 24. Neurosci Biobehav Rev. 2013. PMID: 23707776 Review.
Extensive neuropathological studies have established a compelling link between abnormalities in structure and function of subcortical monoaminergic (MA-ergic) systems and the pathophysiology of Alzheimer's disease (AD). ...This review aims to explore the link between ab …
Extensive neuropathological studies have established a compelling link between abnormalities in structure and function of subcortical …
Biochemical, Biomedical and Metabolic Aspects of Imidazole-Containing Dipeptides with the Inherent Complexity to Neurodegenerative Diseases and Various States of Mental Well-Being: A Challenging Correction and Neurotherapeutic Pharmaceutical Biotechnology for Treating Cognitive Deficits, Depression and Intellectual Disabilities.
Babizhayev MA. Babizhayev MA. Curr Pharm Biotechnol. 2014;15(8):738-78. doi: 10.2174/1389201015666140827104918. Curr Pharm Biotechnol. 2014. PMID: 25158972 Review.
Carnosine and carcinine exhibit a well-documented anti-glycating activity against the glycation of proteins, including low-density lipoproteins, glucose degradation products, esterase, histones. A tissue carnosine-degrading enzyme (CN2) colocalized with the activity of …
Carnosine and carcinine exhibit a well-documented anti-glycating activity against the glycation of proteins, including low-density li …
Mechanism of copper transport from plasma to hepatocytes.
Ettinger MJ, Darwish HM, Schmitt RC. Ettinger MJ, et al. Fed Proc. 1986 Nov;45(12):2800-4. Fed Proc. 1986. PMID: 3533645 Review.
Albumin markedly inhibits copper uptake by a substrate removal mechanism, and histidine facilitates albumin-inhibited copper uptake. The active species that delivers copper to hepatocytes under conditions of excess albumin and excess histidine is the His2Cu complex. …
Albumin markedly inhibits copper uptake by a substrate removal mechanism, and histidine facilitates albumin-inhibited copper uptake. …
Protein electron transfer (mechanism and reproductive toxicity): iminium, hydrogen bonding, homoconjugation, amino acid side chains (redox and charged), and cell signaling.
Kovacic P. Kovacic P. Birth Defects Res C Embryo Today. 2007 Mar;81(1):51-64. doi: 10.1002/bdrc.20086. Birth Defects Res C Embryo Today. 2007. PMID: 17539014 Review.
Redox amino acids (AAs), mainly tyrosine (Tyr), tryptophan (Typ), histidine (His), cysteine (Cys), disulfide, and methionine (Met), are involved in the competing processes for radical formation: direct hydrogen atom abstraction versus electron and proton loss. ...There is …
Redox amino acids (AAs), mainly tyrosine (Tyr), tryptophan (Typ), histidine (His), cysteine (Cys), disulfide, and methionine (Met), a …
Hypokalemic periodic paralysis: an autosomal dominant muscle disorder caused by mutations in a voltage-gated calcium channel.
Lapie P, Lory P, Fontaine B. Lapie P, et al. Neuromuscul Disord. 1997 Jun;7(4):234-40. doi: 10.1016/s0960-8966(97)00435-5. Neuromuscul Disord. 1997. PMID: 9196905 Review.
Mutations affect segments S4 of domains II and IV, changing an arginine in position 528 and 1239 into an histidine, or an histidine or a glycine respectively. Surprisingly, expressing in vitro mutants channels in a non-muscular environment resulted in functional cal …
Mutations affect segments S4 of domains II and IV, changing an arginine in position 528 and 1239 into an histidine, or an histidin …
Are aberrant transcripts of FHIT, TSG101, and PTEN/MMAC1 oncogenesis related?
Wang NM, Chang JG. Wang NM, et al. Int J Mol Med. 1999 May;3(5):491-5. doi: 10.3892/ijmm.3.5.491. Int J Mol Med. 1999. PMID: 10202180 Review.
Tumor suppressor gene mutations in TSG101, FHIT, and PTEN/MMAC1 were found in many types of cancer and the defects in these genes are responsible for the tumor development. ...
Tumor suppressor gene mutations in TSG101, FHIT, and PTEN/MMAC1 were found in many types of cancer and the defects in these genes are …
13 results