Skip to main page content
U.S. flag

An official website of the United States government

Dot gov

The .gov means it’s official.
Federal government websites often end in .gov or .mil. Before sharing sensitive information, make sure you’re on a federal government site.

Https

The site is secure.
The https:// ensures that you are connecting to the official website and that any information you provide is encrypted and transmitted securely.

Access keys NCBI Homepage MyNCBI Homepage Main Content Main Navigation

Search Page

Filters

My NCBI Filters

Results by year

Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1977 2
1978 1
1980 1
1982 5
1985 1
1987 1
1988 7
1989 4
1990 6
1991 8
1992 18
1993 10
1994 12
1995 24
1996 11
1997 20
1998 12
1999 10
2000 14
2001 14
2002 9
2003 12
2004 16
2005 17
2006 21
2007 25
2008 21
2009 12
2010 21
2011 21
2012 21
2013 28
2014 20
2015 35
2016 35
2017 50
2018 37
2019 52
2020 45
2021 51
2022 43
2023 40
2024 12

Text availability

Article attribute

Article type

Publication date

Search Results

748 results

Results by year

Filters applied: . Clear all
Page 1
Interstitial lung diseases.
Wijsenbeek M, Suzuki A, Maher TM. Wijsenbeek M, et al. Lancet. 2022 Sep 3;400(10354):769-786. doi: 10.1016/S0140-6736(22)01052-2. Epub 2022 Aug 11. Lancet. 2022. PMID: 35964592 Review.
Most interstitial lung diseases are characterised by inflammation or fibrosis within the interstitial space, the primary consequence of which is impaired gas exchange, resulting in breathlessness, diminished exercise tolerance, and decreased quality of life. …
Most interstitial lung diseases are characterised by inflammation or fibrosis within the interstitial space, the primar …
Cardiac fibrosis.
Frangogiannis NG. Frangogiannis NG. Cardiovasc Res. 2021 May 25;117(6):1450-1488. doi: 10.1093/cvr/cvaa324. Cardiovasc Res. 2021. PMID: 33135058 Free PMC article. Review.
Myocardial fibrosis, the expansion of the cardiac interstitium through deposition of extracellular matrix proteins, is a common pathophysiologic companion of many different myocardial conditions. ...This review article discusses the molecular mechanisms involved in …
Myocardial fibrosis, the expansion of the cardiac interstitium through deposition of extracellular matrix proteins, is a commo …
Senescence mechanisms and targets in the heart.
Chen MS, Lee RT, Garbern JC. Chen MS, et al. Cardiovasc Res. 2022 Mar 25;118(5):1173-1187. doi: 10.1093/cvr/cvab161. Cardiovasc Res. 2022. PMID: 33963378 Free PMC article. Review.
Cellular senescence is a state of irreversible cell cycle arrest associated with ageing. Senescence of different cardiac cell types can direct the pathophysiology of cardiovascular diseases (CVDs) such as atherosclerosis, myocardial infarction, and cardiac fibros …
Cellular senescence is a state of irreversible cell cycle arrest associated with ageing. Senescence of different cardiac cell types c …
Cardiac sarcoidosis: diagnosis and management.
Markatis E, Afthinos A, Antonakis E, Papanikolaou IC. Markatis E, et al. Rev Cardiovasc Med. 2020 Sep 30;21(3):321-338. doi: 10.31083/j.rcm.2020.03.102. Rev Cardiovasc Med. 2020. PMID: 33070538 Free article. Review.
End-organ disease consists of granulomatous inflammation, which if left untreated or not resolved spontaneously, leads to permanent fibrosis and end-organ dysfunction. Cardiac involvement and fibrosis in sarcoidosis occur in 5-10% of cases and is becoming inc …
End-organ disease consists of granulomatous inflammation, which if left untreated or not resolved spontaneously, leads to permanent fibro …
Progressive pulmonary fibrosis: an expert group consensus statement.
Rajan SK, Cottin V, Dhar R, Danoff S, Flaherty KR, Brown KK, Mohan A, Renzoni E, Mohan M, Udwadia Z, Shenoy P, Currow D, Devraj A, Jankharia B, Kulshrestha R, Jones S, Ravaglia C, Quadrelli S, Iyer R, Dhooria S, Kolb M, Wells AU. Rajan SK, et al. Eur Respir J. 2023 Mar 30;61(3):2103187. doi: 10.1183/13993003.03187-2021. Print 2023 Mar. Eur Respir J. 2023. PMID: 36517177 Free PMC article. Review.
This expert group consensus statement emphasises the need for standardising the definition of progressive fibrosing interstitial lung diseases (F-ILDs), with an accurate initial diagnosis being of paramount importance in ensuring appropriate initial management. ...I …
This expert group consensus statement emphasises the need for standardising the definition of progressive fibrosing interstitial …
Myocardial Interstitial Fibrosis in Heart Failure: Biological and Translational Perspectives.
González A, Schelbert EB, Díez J, Butler J. González A, et al. J Am Coll Cardiol. 2018 Apr 17;71(15):1696-1706. doi: 10.1016/j.jacc.2018.02.021. J Am Coll Cardiol. 2018. PMID: 29650126 Free article. Review.
Myocardial interstitial fibrosis contributes to left ventricular dysfunction leading to the development of heart failure. ...This is partly related to the paucity of biomarkers to accurately identify myocardial interstitial fibrosis and to the l …
Myocardial interstitial fibrosis contributes to left ventricular dysfunction leading to the development of heart failur …
Extracellular matrix remodeling and cardiac fibrosis.
Li L, Zhao Q, Kong W. Li L, et al. Matrix Biol. 2018 Aug;68-69:490-506. doi: 10.1016/j.matbio.2018.01.013. Epub 2018 Jan 31. Matrix Biol. 2018. PMID: 29371055 Review.
Cardiac fibrosis, characterized by excessive deposition of extracellular matrix (ECM) proteins in the myocardium, distorts the architecture of the myocardium, facilitates the progression of arrhythmia and cardiac dysfunction, and influences the clinical cours …
Cardiac fibrosis, characterized by excessive deposition of extracellular matrix (ECM) proteins in the myocardium, distorts the …
Cardiac fibrosis: Cell biological mechanisms, molecular pathways and therapeutic opportunities.
Frangogiannis NG. Frangogiannis NG. Mol Aspects Med. 2019 Feb;65:70-99. doi: 10.1016/j.mam.2018.07.001. Epub 2018 Aug 2. Mol Aspects Med. 2019. PMID: 30056242 Review.
Activated myofibroblasts are the main effector cells in cardiac fibrosis; their expansion following myocardial injury is primarily driven through activation of resident interstitial cell populations. ...Due to the reparative nature of many forms of cardiac …
Activated myofibroblasts are the main effector cells in cardiac fibrosis; their expansion following myocardial injury is prima …
Calcific aortic valve disease: mechanisms, prevention and treatment.
Moncla LM, Briend M, Bossé Y, Mathieu P. Moncla LM, et al. Nat Rev Cardiol. 2023 Aug;20(8):546-559. doi: 10.1038/s41569-023-00845-7. Epub 2023 Feb 24. Nat Rev Cardiol. 2023. PMID: 36829083 Review.
Calcific aortic valve disease (CAVD) is the most common disorder affecting heart valves and is characterized by thickening, fibrosis and mineralization of the aortic valve leaflets. Analyses of surgically explanted aortic valve leaflets have shown that dystrophic mi …
Calcific aortic valve disease (CAVD) is the most common disorder affecting heart valves and is characterized by thickening, fibros …
Mitochondrial dynamics, mitophagy and cardiovascular disease.
Vásquez-Trincado C, García-Carvajal I, Pennanen C, Parra V, Hill JA, Rothermel BA, Lavandero S. Vásquez-Trincado C, et al. J Physiol. 2016 Feb 1;594(3):509-25. doi: 10.1113/JP271301. Epub 2016 Jan 15. J Physiol. 2016. PMID: 26537557 Free PMC article. Review.
Cardiac hypertrophy is often initiated as an adaptive response to haemodynamic stress or myocardial injury, and allows the heart to meet an increased demand for oxygen. Although initially beneficial, hypertrophy can ultimately contribute to the progression of car …
Cardiac hypertrophy is often initiated as an adaptive response to haemodynamic stress or myocardial injury, and allows the heart …
748 results