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1973
2024

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Year Number of Results
1973 1
1979 2
1982 1
1983 1
1984 2
1985 1
1986 1
1998 1
2000 1
2024 0

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11 results

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Page 1
Regulation of macrophage phagocytosis.
Coleman DL. Coleman DL. Eur J Clin Microbiol. 1986 Feb;5(1):1-5. doi: 10.1007/BF02013451. Eur J Clin Microbiol. 1986. PMID: 3084238 Review.
A number of diseases are caused by qualitative or quantitative disorders of phagocytosis by four major mechanisms: a decrease in the flow of blood to organs which contain macrophages (e.g. congestive heart failure and portal hypertension); a decrease in the quantity of tissue whi …
A number of diseases are caused by qualitative or quantitative disorders of phagocytosis by four major mechanisms: a decrease in the flow of …
[Tuftsin].
Mücke D. Mücke D. Allerg Immunol (Leipz). 1984;30(3):127-38. Allerg Immunol (Leipz). 1984. PMID: 6209973 Review. German.
The action of tuftsin is mediated by specific receptor sites on the surface of these cells. Its antitumor activity has been shown in vitro as well as in vivo. ...--The feature of tuftsin and its low toxicity make it a useful agent for immunotherapy....
The action of tuftsin is mediated by specific receptor sites on the surface of these cells. Its antitumor activity has been shown in …
The clinical and physiological aspects of tuftsin deficiency syndromes exhibiting defective phagocytosis.
Najjar VA. Najjar VA. Klin Wochenschr. 1979 Aug 1;57(15):751-6. doi: 10.1007/BF01478032. Klin Wochenschr. 1979. PMID: 385971 Review.
One of these is in the spleen and the other on the outer membrane of the phagocyte. Congenital tuftsin deficiency usually arises when the peptide is mutated to an inactive peptide. The acquired type occurs if the spleen function is curtailed by removal or disease. …
One of these is in the spleen and the other on the outer membrane of the phagocyte. Congenital tuftsin deficiency usually aris …
Tuftsin, a natural activator of phagocyte cells: an overview.
Najjar VA. Najjar VA. Ann N Y Acad Sci. 1983;419:1-11. doi: 10.1111/j.1749-6632.1983.tb37086.x. Ann N Y Acad Sci. 1983. PMID: 6370072 Review.
The oligopeptide was isolated, its structure determined (Thr-Lys-Pro-Arg) and synthesized. The discovery of human mutants with tuftsin deficiency exhibiting signs and symptoms of frequent severe infection further emphasized the specific biological function of the te …
The oligopeptide was isolated, its structure determined (Thr-Lys-Pro-Arg) and synthesized. The discovery of human mutants with tuftsin
Recent developments in immunomodulatory therapy.
Bardana EJ Jr. Bardana EJ Jr. J Allergy Clin Immunol. 1985 Apr;75(4):423-40. doi: 10.1016/s0091-6749(85)80011-7. J Allergy Clin Immunol. 1985. PMID: 2579994 Review. No abstract available.
Immune function of the spleen.
Trigg ME. Trigg ME. South Med J. 1979 May;72(5):593-9. doi: 10.1097/00007611-197905000-00025. South Med J. 1979. PMID: 375402 Review. No abstract available.
[Tuftsin deficiency].
Kohsaka T. Kohsaka T. Ryoikibetsu Shokogun Shirizu. 2000;(32):153-6. Ryoikibetsu Shokogun Shirizu. 2000. PMID: 11212673 Review. Japanese. No abstract available.
[Tuftsin deficiency].
Souillet G. Souillet G. Pediatrie. 1973 Mar;28(2):223-7. Pediatrie. 1973. PMID: 4125356 Review. French. No abstract available.
[Congenital familial tuftsin deficiency].
Otsuka T, Niho Y. Otsuka T, et al. Ryoikibetsu Shokogun Shirizu. 1998;(21 Pt 2):67-9. Ryoikibetsu Shokogun Shirizu. 1998. PMID: 9833430 Review. Japanese. No abstract available.
Cytophilic gamma-globulin and tuftsin.
Najjar VA. Najjar VA. Surv Immunol Res. 1982;1(1):9-16. doi: 10.1007/BF02918236. Surv Immunol Res. 1982. PMID: 6195710 Review. No abstract available.
11 results