|
| Status |
Public on Feb 26, 2019 |
| Title |
PTENα/β paradoxically promote carcinogenesis through WDR5-H3K4me3 axis |
| Organism |
Homo sapiens |
| Experiment type |
Genome binding/occupancy profiling by high throughput sequencing
Expression profiling by high throughput sequencing
|
| Summary |
This SuperSeries is composed of the SubSeries listed below.
|
| |
|
| Overall design |
Refer to individual Series
|
| |
|
| Citation(s) |
31685992 |
| Submission date |
Feb 24, 2019 |
| Last update date |
Nov 14, 2019 |
| Contact name |
ge mengkai |
| E-mail(s) |
gemengk@sjtu.edu.cn
|
| Phone |
15201867201
|
| Organization name |
Shanghai Jiao Tong University School of Medicine (SJTU-SM)
|
| Street address |
shanghai
|
| City |
shanghai |
| State/province |
State... |
| ZIP/Postal code |
13185541251gmk |
| Country |
China |
| |
|
| Platforms (1) |
| GPL24676 |
Illumina NovaSeq 6000 (Homo sapiens) |
|
| Samples (21)
|
|
| This SuperSeries is composed of the following SubSeries:
|
| GSE126982 |
PTENα/β paradoxically promote carcinogenesis through WDR5-H3K4me3 axis [ChIP-seq] |
| GSE126983 |
PTENα/β paradoxically promote carcinogenesis through WDR5-H3K4me3 axis [RNA-seq] |
| GSE135477 |
PTENa/b paradoxically promote carcinogenesis through WDR5-H3K4me3 axis [RNA-seq 2] |
| GSE135478 |
PTENa/b paradoxically promote carcinogenesis through WDR5-H3K4me3 axis [ChIP-seq 2] |
|
| Relations |
| BioProject |
PRJNA524001 |
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