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Status |
Public on Dec 04, 2020 |
Title |
Fat1 deletion promotes hybrid EMT state with enhanced tumor progression, stemness, and metastasis |
Organisms |
Homo sapiens; Mus musculus |
Experiment type |
Expression profiling by high throughput sequencing Genome binding/occupancy profiling by high throughput sequencing Other
|
Summary |
This SuperSeries is composed of the SubSeries listed below.
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|
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Overall design |
Refer to individual Series
|
|
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Citation(s) |
33328637 |
|
Submission date |
Sep 24, 2020 |
Last update date |
Mar 05, 2021 |
Contact name |
Yura Song |
E-mail(s) |
yura.song@ulb.be
|
Organization name |
Université Libre de Bruxelles
|
Lab |
Laboratory of Stem Cells and Cancer
|
Street address |
808, route de Lennik
|
City |
Bruxelles |
ZIP/Postal code |
1070 |
Country |
Belgium |
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Platforms (2) |
GPL24247 |
Illumina NovaSeq 6000 (Mus musculus) |
GPL24676 |
Illumina NovaSeq 6000 (Homo sapiens) |
|
Samples (62)
|
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This SuperSeries is composed of the following SubSeries:
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GSE158500 |
Fat1 deletion promotes hybrid EMT state with enhanced tumor progression, stemness, and metastasis (ATAC-Seq) |
GSE158501 |
Fat1 deletion promotes hybrid EMT state with enhanced tumor progression, stemness, and metastasis (human RNA-Seq) |
GSE158502 |
Fat1 deletion promotes hybrid EMT state with enhanced tumor progression, stemness, and metastasis (mouse RNA-Seq) |
GSE158503 |
Fat1 deletion promotes hybrid EMT state with enhanced tumor progression, stemness, and metastasis (exome) |
GSE158505 |
Fat1 deletion promotes hybrid EMT state with enhanced tumor progression, stemness, and metastasis (DNA-Seq) |
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Relations |
BioProject |
PRJNA665481 |