|
Status |
Public on Feb 04, 2022 |
Title |
CRISPR activation screen identifies BCL-2 proteins and B3GNT2 as drivers of cancer resistance to T cell-mediated cytotoxicity |
Organism |
Homo sapiens |
Experiment type |
Genome binding/occupancy profiling by high throughput sequencing Expression profiling by high throughput sequencing
|
Summary |
This SuperSeries is composed of the SubSeries listed below.
|
|
|
Overall design |
Refer to individual Series
|
|
|
Citation(s) |
35338135 |
|
Submission date |
Oct 19, 2020 |
Last update date |
Apr 27, 2022 |
Contact name |
Julia Joung |
E-mail(s) |
julia@joung.science
|
Organization name |
Broad Institute
|
Street address |
75 Ames St
|
City |
Cambridge |
State/province |
MA |
ZIP/Postal code |
02142 |
Country |
USA |
|
|
Platforms (1) |
GPL18573 |
Illumina NextSeq 500 (Homo sapiens) |
|
Samples (37)
|
|
This SuperSeries is composed of the following SubSeries: |
GSE159538 |
CRISPR activation screen identifies BCL-2 proteins and B3GNT2 as drivers of cancer resistance to T cell-mediated cytotoxicity |
GSE159539 |
CRISPR activation screen identifies BCL-2 proteins and B3GNT2 as drivers of cancer resistance to T cell-mediated cytotoxicity |
GSE197309 |
CRISPR activation screen identifies BCL-2 proteins and B3GNT2 as drivers of cancer resistance to T cell-mediated cytotoxicity (sgRNA distribution) |
|
Relations |
BioProject |
PRJNA669676 |