|
Status |
Public on Dec 13, 2023 |
Title |
BPTF inhibitor increases gastric cancer response to Erlotinib by epigenetically regulating c-MYC/PLCG1/pErk axis. |
Organism |
Homo sapiens |
Experiment type |
Genome binding/occupancy profiling by high throughput sequencing Expression profiling by high throughput sequencing
|
Summary |
This SuperSeries is composed of the SubSeries listed below.
|
|
|
Overall design |
Refer to individual Series
|
|
|
Citation(s) |
37863665 |
|
Submission date |
Apr 05, 2022 |
Last update date |
Dec 14, 2023 |
Contact name |
Li Fangyuan |
E-mail(s) |
lifangyuan13@icloud.com
|
Phone |
18701877783
|
Organization name |
Shanghai Jiao Tong University
|
Street address |
Ruijin Er road
|
City |
Shanghai |
State/province |
Shanghai |
ZIP/Postal code |
021000 |
Country |
China |
|
|
Platforms (2) |
GPL10999 |
Illumina Genome Analyzer IIx (Homo sapiens) |
GPL24676 |
Illumina NovaSeq 6000 (Homo sapiens) |
|
Samples (6)
|
|
This SuperSeries is composed of the following SubSeries: |
GSE200181 |
BPTF inhibitor increases gastric cancer response to Erlotinib by epigenetically regulating c-MYC/PLCG1/pErk axis [ChIP-seq] |
GSE200182 |
BPTF inhibitor increases gastric cancer response to Erlotinib by epigenetically regulating c-MYC/PLCG1/pErk axis [RNA-seq] |
|
Relations |
BioProject |
PRJNA823552 |