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Status |
Public on Jan 13, 2023 |
Title |
Loss of CPSF6 causes developmental disease via bimodal changes in polyadenylation site usage and protein expression |
Organisms |
Danio rerio; Homo sapiens |
Experiment type |
Other
|
Summary |
This SuperSeries is composed of the SubSeries listed below.
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Overall design |
Refer to individual Series
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Citation(s) |
- de Prisco N, Ford C, Elrod ND, Lee W et al. Alternative polyadenylation alters protein dosage by switching between intronic and 3'UTR sites. Sci Adv 2023 Feb 17;9(7):eade4814. PMID: 36800428
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Submission date |
Jun 21, 2022 |
Last update date |
Feb 23, 2023 |
Contact name |
Hari Krishna Yalamanchili |
E-mail(s) |
hari.yalamanchili@bcm.edu
|
Organization name |
Baylor College of Medicine
|
Street address |
1250 Moursund St #1125,
|
City |
Houston, |
State/province |
TX |
ZIP/Postal code |
77030 |
Country |
USA |
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Platforms (2) |
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Samples (29)
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This SuperSeries is composed of the following SubSeries: |
GSE206557 |
Loss of CPSF6 causes developmental disease via bimodal changes in polyadenylation site usage and protein expression [Human] |
GSE206558 |
Loss of CPSF6 causes developmental disease via bimodal changes in polyadenylation site usage and protein expression [Zebrafish] |
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Relations |
BioProject |
PRJNA851372 |