|
| Status |
Public on Jun 14, 2023 |
| Title |
Decitabine disrupts EBV genomic epiallele DNA methylation patterns around CTCF binding sites to increase chromatin accessibility and lytic transcription in gastric cancer |
| Organism |
human gammaherpesvirus 4 |
| Experiment type |
Methylation profiling by high throughput sequencing
Genome binding/occupancy profiling by high throughput sequencing
|
| Summary |
This SuperSeries is composed of the SubSeries listed below.
|
| |
|
| Overall design |
Refer to individual Series
|
| |
|
| Citation(s) |
37606370, 37889078 |
| |
| Submission date |
Jun 12, 2023 |
| Last update date |
Dec 22, 2023 |
| Contact name |
Sarah Alp |
| E-mail(s) |
salp@wistar.org
|
| Organization name |
The Wistar Institute
|
| Street address |
3601 Spruce Street
|
| City |
Philadelphia |
| State/province |
PA |
| ZIP/Postal code |
19401 |
| Country |
USA |
| |
|
| Platforms (2) |
| GPL25190 |
Illumina HiSeq 2500 (Human gammaherpesvirus 4) |
| GPL33481 |
Illumina NextSeq 500 (Human gammaherpesvirus 4) |
|
| Samples (16)
|
|
| This SuperSeries is composed of the following SubSeries: |
| GSE233330 |
Decitabine disrupts EBV genomic epiallele DNA methylation patterns around CTCF binding sites to increase chromatin accessibility and lytic transcription in gastric cancer [RRBS] |
| GSE234603 |
Decitabine disrupts EBV genomic epiallele DNA methylation patterns around CTCF binding sites to increase chromatin accessibility and lytic transcription in gastric cancer [ChIP-seq] |
|
| Relations |
| BioProject |
PRJNA982703 |
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