|
| Status |
Public on May 29, 2024 |
| Title |
Dysregulated innate immune signaling cooperates with RUNX1 mutations to transform an MDS-like disease to AML |
| Organism |
Mus musculus |
| Experiment type |
Expression profiling by high throughput sequencing
Other
|
| Summary |
This SuperSeries is composed of the SubSeries listed below.
|
| |
|
| Overall design |
Refer to individual Series
|
| |
|
| Citation(s) |
38784013 |
| |
| Submission date |
Apr 15, 2024 |
| Last update date |
May 30, 2024 |
| Contact name |
Kwangmin Choi |
| Organization name |
Cincinnati Children's Hospital Medical Center
|
| Department |
Experimental Hematology & Cancer Biology
|
| Street address |
333 Burnet Avenue
|
| City |
Cincinnati |
| State/province |
Ohio |
| ZIP/Postal code |
45229 |
| Country |
USA |
| |
|
| Platforms (2) |
| GPL13112 |
Illumina HiSeq 2000 (Mus musculus) |
| GPL17021 |
Illumina HiSeq 2500 (Mus musculus) |
|
| Samples (24)
|
|
| This SuperSeries is composed of the following SubSeries: |
| GSE264051 |
Dysregulated innate immune signaling cooperates with RUNX1 mutations to transform an MDS-like disease to AML (RNA-Seq) |
| GSE264052 |
Dysregulated innate immune signaling cooperates with RUNX1 mutations to transform an MDS-like disease to AML (WES) |
|
| Relations |
| BioProject |
PRJNA1100608 |
Less...
More...