|
Status |
Public on Apr 28, 2017 |
Title |
Epigenetic identity in AML is mostly dependent on disruption of non-promoter regulatory elements and reveals potentially antagonistic effects of mutations in epigenetic modifiers |
Organism |
Homo sapiens |
Experiment type |
Third-party reanalysis Methylation profiling by high throughput sequencing
|
Summary |
This SuperSeries is composed of the SubSeries listed below.
|
|
|
Overall design |
Refer to individual Series
|
|
|
Citation(s) |
28408400 |
|
Submission date |
Apr 28, 2017 |
Last update date |
May 15, 2019 |
Contact name |
Jacob Lowell Glass |
Organization name |
Memorial Sloan Kettering Cancer Center
|
Street address |
1275 York Avenue
|
City |
New York |
State/province |
NY |
ZIP/Postal code |
10065 |
Country |
USA |
|
|
Platforms (1) |
GPL11154 |
Illumina HiSeq 2000 (Homo sapiens) |
|
Samples (126)
|
|
This SuperSeries is composed of the following SubSeries: |
GSE86952 |
Epigenetic identity in AML is mostly dependent on disruption of non-promoter regulatory elements and reveals potentially antagonistic effects of mutations in epigenetic modifiers [human] |
GSE96744 |
Epigenetic identity in AML is mostly dependent on disruption of non-promoter regulatory elements and reveals potentially antagonistic effects of mutations in epigenetic modifiers [mouse] |
|
Relations |
BioProject |
PRJNA384795 |