Analysis of BEAS-2B lung epithelial cells treated with 2.5 uM arsenic trioxide for 6 months. Chronic arsenic exposure increases lung cancer risk. Results provide insight into the molecular mechanisms underlying lung carcinogenesis associated with arsenic exposure.
GPL6480:
Agilent-014850 Whole Human Genome Microarray 4x44K G4112F (Probe Name version)
Citation:
Stueckle TA, Lu Y, Davis ME, Wang L et al. Chronic occupational exposure to arsenic induces carcinogenic gene signaling networks and neoplastic transformation in human lung epithelial cells. Toxicol Appl Pharmacol 2012 Jun 1;261(2):204-16. PMID: 22521957