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Status |
Public on Jun 23, 2018 |
Title |
KCa3.1 depletion effect on hepatocellular carcinoma cell line Huh7 |
Organism |
Homo sapiens |
Experiment type |
Expression profiling by array
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Summary |
Analysis of Huh7 hepatocellular carcinoma (HCC) cells depleted for potassium channel KCa3.1, the intermediate conductance calcium-activated potassium channel. Result provide insight into the role of KCa3.1 in the carcinogenesis of HCC. We used microarrays to detail the gene expression differences between KCa3.1 knockdown and negative control in Huh7 HCC cells.
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Overall design |
A human gene expression chip was applied to assess the gene transcriptional profiles of KCa3.1 silenced Huh7 cells and Huh7 control cells.
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Contributor(s) |
Song P, Du Y |
Citation missing |
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Submission date |
Jun 22, 2018 |
Last update date |
Aug 23, 2018 |
Contact name |
Yehui Du |
E-mail(s) |
670354994@qq.com
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Phone |
+8615267470578
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Organization name |
Zhejiang University
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Street address |
79# Qinchun Road
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City |
Hangzhou |
State/province |
Zhejiang |
ZIP/Postal code |
310000 |
Country |
China |
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Platforms (1) |
GPL15207 |
[PrimeView] Affymetrix Human Gene Expression Array |
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Samples (6)
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Relations |
BioProject |
PRJNA477558 |