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Status |
Public on Oct 19, 2022 |
Title |
The histone H3G34R mutation disrupts the epigenome via catalytic inactivation of the ASH1 H3K36 methyltransferase |
Organism |
Neurospora crassa |
Experiment type |
Expression profiling by high throughput sequencing Genome binding/occupancy profiling by high throughput sequencing Methylation profiling by high throughput sequencing
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Summary |
This SuperSeries is composed of the SubSeries listed below.
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Overall design |
Refer to individual Series
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Citation missing |
Has this study been published? Please login to update or notify GEO. |
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Submission date |
Jul 17, 2019 |
Last update date |
Oct 22, 2022 |
Contact name |
Elizabeth Toomey Wiles |
E-mail(s) |
tishw@uoregon.edu
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Organization name |
University of Oregon
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Department |
Biology, Institute of Molecular Biology
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Lab |
Selker
|
Street address |
1229 University of Oregon; 1318 Franklin Blvd.
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City |
Eugene |
State/province |
OR |
ZIP/Postal code |
97403 |
Country |
USA |
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Platforms (3) |
GPL16164 |
Illumina HiSeq 2000 (Neurospora crassa) |
GPL20660 |
Illumina NextSeq 500 (Neurospora crassa) |
GPL23150 |
Illumina HiSeq 4000 (Neurospora crassa) |
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Samples (20)
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This SuperSeries is composed of the following SubSeries: |
GSE134445 |
The histone H3G34R mutation disrupts the epigenome via catalytic inactivation of the ASH1 H3K36 methyltransferase [ATAC-seq] |
GSE134449 |
The histone H3G34R mutation disrupts the epigenome via catalytic inactivation of the ASH1 H3K36 methyltransferase [ChIP-seq] |
GSE134451 |
The histone H3G34R mutation disrupts the epigenome via catalytic inactivation of the ASH1 H3K36 methyltransferase [RNA-seq] |
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Relations |
BioProject |
PRJNA555141 |