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Series GSE211445 Query DataSets for GSE211445
Status Public on Jul 19, 2023
Title C99R mutation in IRF4 drives a novel gain of function binding and gene upregulation in classical Hodgkin lymphoma
Organism Homo sapiens
Experiment type Expression profiling by high throughput sequencing
Genome binding/occupancy profiling by high throughput sequencing
Expression profiling by array
Summary This SuperSeries is composed of the SubSeries listed below.
 
Overall design Refer to individual Series
 
Citation(s) 37935654
Submission date Aug 17, 2022
Last update date Dec 01, 2023
Contact name Pierre Daniel Cauchy
E-mail(s) cauchy@ie-freiburg.mpg.de
Phone +49 (0)761 270-77576
Organization name University Medical Center Freiburg
Department Zentrum für Translationale Zellforschung
Lab AG Onco-Immunology
Street address Breisacher Str. 115
City Freiburg
State/province Baden-Württemberg
ZIP/Postal code 79106
Country Germany
 
Platforms (3)
GPL10558 Illumina HumanHT-12 V4.0 expression beadchip
GPL11154 Illumina HiSeq 2000 (Homo sapiens)
GPL13393 AB SOLiD 4 System (Homo sapiens)
Samples (63)
GSM6470185 Reh_DHS_1
GSM6470186 Reh_DHS_2
GSM6470187 Reh_DHS_3
This SuperSeries is composed of the following SubSeries:
GSE211441 C99R mutation in IRF4 drives a novel gain of function binding and gene upregulation in classical Hodgkin lymphoma [DNase-Seq]
GSE211443 C99R mutation in IRF4 drives a novel gain of function binding and gene upregulation in classical Hodgkin lymphoma [ChIP-Seq]
GSE211444 C99R mutation in IRF4 drives a novel gain of function binding and gene upregulation in classical Hodgkin lymphoma [RNA-Seq]
Relations
BioProject PRJNA870420

Download family Format
SOFT formatted family file(s) SOFTHelp
MINiML formatted family file(s) MINiMLHelp
Series Matrix File(s) TXTHelp

Supplementary file Size Download File type/resource
GSE211445_RAW.tar 75.8 Mb (http)(custom) TAR (of IDAT)
SRA Run SelectorHelp

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