|
Status |
Public on Jul 19, 2023 |
Title |
C99R mutation in IRF4 drives a novel gain of function binding and gene upregulation in classical Hodgkin lymphoma |
Organism |
Homo sapiens |
Experiment type |
Expression profiling by high throughput sequencing Genome binding/occupancy profiling by high throughput sequencing Expression profiling by array
|
Summary |
This SuperSeries is composed of the SubSeries listed below.
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|
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Overall design |
Refer to individual Series
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|
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Citation(s) |
37935654 |
|
Submission date |
Aug 17, 2022 |
Last update date |
Dec 01, 2023 |
Contact name |
Pierre Daniel Cauchy |
E-mail(s) |
cauchy@ie-freiburg.mpg.de
|
Phone |
+49 (0)761 270-77576
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Organization name |
University Medical Center Freiburg
|
Department |
Zentrum für Translationale Zellforschung
|
Lab |
AG Onco-Immunology
|
Street address |
Breisacher Str. 115
|
City |
Freiburg |
State/province |
Baden-Württemberg |
ZIP/Postal code |
79106 |
Country |
Germany |
|
|
Platforms (3) |
GPL10558 |
Illumina HumanHT-12 V4.0 expression beadchip |
GPL11154 |
Illumina HiSeq 2000 (Homo sapiens) |
GPL13393 |
AB SOLiD 4 System (Homo sapiens) |
|
Samples (63)
|
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This SuperSeries is composed of the following SubSeries:
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GSE211441 |
C99R mutation in IRF4 drives a novel gain of function binding and gene upregulation in classical Hodgkin lymphoma [DNase-Seq] |
GSE211443 |
C99R mutation in IRF4 drives a novel gain of function binding and gene upregulation in classical Hodgkin lymphoma [ChIP-Seq] |
GSE211444 |
C99R mutation in IRF4 drives a novel gain of function binding and gene upregulation in classical Hodgkin lymphoma [RNA-Seq] |
GSE211913 |
C99R mutation in IRF4 drives a novel gain of function binding and gene upregulation in classical Hodgkin lymphoma [Illumina BeadChip] |
|
Relations |
BioProject |
PRJNA870420 |