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Items: 3

1.

Senescence induced by BMI1 inhibition is a therapeutic vulnerability in H3K27M-mutant DIPG

(Submitter supplied) We studied the effect of BMI1 inhibition in diffuse intrinsic pontine glioma cells (DIPG). We reported the application of chromatin immunoprecipitation sequencing for high-thoughput profiling of BMI1 and H2AK119ub in diffuse intrinsic pontine glioma cells (DIPG) in response to BMI1 inhibition. We also performed bulk RNA sequencing of DIPG cells in response to pharmacological inhibition or knockdown of BMI1. more...
Organism:
Homo sapiens
Type:
Genome binding/occupancy profiling by high throughput sequencing; Expression profiling by high throughput sequencing
Platforms:
GPL24676 GPL16791 GPL27644
30 Samples
Download data: TDF, TXT
Series
Accession:
GSE140768
ID:
200140768

PubMed Full text in PMC Similar studies Analyze with GEO2RSRA Run Selector

2.

Illumina Novaseq 6000 (Homo sapiens)

Organism:
Homo sapiens
26 Series
680 Samples
Download data
Platform
Accession:
GPL27644
ID:
100027644

3.

H2AK119_ChIP_SF8628_PTC028

Organism:
Homo sapiens
Source name:
diffuse intrinsic pontine glioma
Platform:
GPL27644
Series:
GSE140768
Download data: TDF
Sample
Accession:
GSM4183387
ID:
304183387

SRA Run Selector

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db=gds|term=GSM4183387[Accession]|query=1|qty=2|blobid=MCID_66dab1f7b8e5607ab868ed35|ismultiple=true|min_list=5|max_list=20|def_tree=20|def_list=|def_view=|url=/Taxonomy/backend/subset.cgi?|trace_url=/stat?
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