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Items: 4

1.

Let-7 miRNAs repress HIC2 to regulate BCL11A transcription and hemoglobin switching [ChIP-Seq]

(Submitter supplied) The fetal hemoglobin (HBG) regulator BCL11A is repressed by HIC2 to regulate hemoglobin switching. HIC2 itself is under developmental control, highly expressed in fetal tissues and decreased in adult cells, but the mechanism of this developmental regulation is unclear. Here, we demonstrate that HIC2 is developmentally controlled by miRNAs, and loss of global miRNA biogenesis through DICER1 depletion leads to upregulation of HIC2 and HBG. more...
Organism:
Homo sapiens
Type:
Genome binding/occupancy profiling by high throughput sequencing
Platform:
GPL30173
10 Samples
Download data: BIGWIG
Series
Accession:
GSE216195
ID:
200216195

PubMed Similar studies

2.

Let-7 miRNAs repress HIC2 to regulate BCL11A transcription and hemoglobin switching

(Submitter supplied) This SuperSeries is composed of the SubSeries listed below.
Organism:
Homo sapiens
Type:
Expression profiling by high throughput sequencing; Genome binding/occupancy profiling by high throughput sequencing; Other
Platform:
GPL30173
26 Samples
Download data: BIGWIG, TSV
Series
Accession:
GSE216197
ID:
200216197

PubMed Similar studies

3.

NextSeq 2000 (Homo sapiens)

Organism:
Homo sapiens
877 Series
16080 Samples
Download data
Platform
Accession:
GPL30173
ID:
100030173

4.

Scramble control GATA1 ChIP-seq rep1

Organism:
Homo sapiens
Source name:
Human erythroblasts
Platform:
GPL30173
Series:
GSE216195 GSE216197
Download data: BIGWIG
Sample
Accession:
GSM6661430
ID:
306661430

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db=gds|term=GSM6661430[Accession]|query=1|qty=2|blobid=MCID_665298ece795547e5f6eb1ba|ismultiple=true|min_list=5|max_list=20|def_tree=20|def_list=|def_view=|url=/Taxonomy/backend/subset.cgi?|trace_url=/stat?
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