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Items: 4

1.

STAT3 inhibition permits epigenetic reprogramming and TLR9/IRF8-driven differentiation of inv(16) acute myeloid leukemia.

(Submitter supplied) This SuperSeries is composed of the SubSeries listed below.
Organism:
Mus musculus
Type:
Expression profiling by high throughput sequencing; Methylation profiling by high throughput sequencing
Platforms:
GPL24247 GPL17021 GPL21103
41 Samples
Download data: MTX, TSV, TXT
Series
Accession:
GSE233099
ID:
200233099

2.

STAT3 inhibition permits epigenetic reprogramming and TLR9/IRF8-driven differentiation of inv(16) acute myeloid leukemia [RNASEQ_AML_DECSCR]

(Submitter supplied) Cultured acute myeloid leukemia (AML) blasts can differentiate into antigen-presenting cells but achieving leukemic cell immunogenicity proved challenging in clinical setting. Despite expressing multiple immunostimulatory receptors, such as Toll-like Receptor 9 (TLR9), AML cells resist immunostimulation. We previously demonstrated that eliminating Signal Transducer and Activator of Transcription 1 and 3 (STAT1/3) signaling in AML cells using decoy DNA strategy (CpG-STAT3dODN) synergized with TLR9 stimulation and resulted in T cell-mediated leukemia regression. more...
Organism:
Mus musculus
Type:
Expression profiling by high throughput sequencing
Platform:
GPL17021
12 Samples
Download data: TXT
Series
Accession:
GSE233096
ID:
200233096

3.

Illumina HiSeq 2500 (Mus musculus)

Organism:
Mus musculus
11102 Series
299258 Samples
Download data
Platform
Accession:
GPL17021
ID:
100017021

4.

DEC_rep2

Organism:
Mus musculus
Source name:
CMM
Platform:
GPL17021
Series:
GSE233096 GSE233099
Download data: TXT
Sample
Accession:
GSM7401379
ID:
307401379

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