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Items: 3

1.

Gene expression changes in Dhcr7 knockout brain, lung and liver at E18.5 compared to wild-type embryos

(Submitter supplied) The genetic defect underlying the human Smith-Lemli-Opitz dysmorphological disorder is loss-of-function mutations affecting the cholesterol synthesis enzyme dehydrocholesterol delta7 reductase, DHCR7. Dhcr7 knockout mice recapitulate the biochemical characteristics, but all knockout pups die within 14h of birth. Tissues of knockout mice accumulate the precursor sterol, 7-dehydrocholesterol, and show reduced levels of cholesterol (J. more...
Organism:
Mus musculus
Type:
Expression profiling by array
Platform:
GPL8063
18 Samples
Download data: TXT
Series
Accession:
GSE39811
ID:
200039811

Analyze with GEO2R

2.

GE Healthcare/Amersham Biosciences CodeLink™ Mouse Whole Genome Bioarray

(Submitter supplied) http://www4.amershambiosciences.com/aptrix/upp01077.nsf/Content/codelink_user_protocols
Organism:
Mus musculus
9 Series
74 Samples
Download data
Platform
Accession:
GPL8063
ID:
100008063

3.

Wild-type liver, F295

Organism:
Mus musculus
Source name:
Liver, wild-type
Platform:
GPL8063
Series:
GSE39811
Download data: TXT
Sample
Accession:
GSM979621
ID:
300979621

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db=gds|term=GSM979621[Accession]|query=1|qty=2|blobid=MCID_666c2d00909a3d46ad8e995a|ismultiple=true|min_list=5|max_list=20|def_tree=20|def_list=|def_view=|url=/Taxonomy/backend/subset.cgi?|trace_url=/stat?
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