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The SaeR/S Gene Regulatory System is Essential for Innate Immune Evasion by Staphylococcus aureus
PubMed Full text in PMC Similar studies Analyze with GEO2R
Analysis of Staphylococcus aureus transcriptome changes in response to inhibition of Rho activity by bicyclomycin
S. aureus gene expression during growth phase and/or S. aureus–PMN interaction
PubMed Similar studies Analyze with GEO2R
NIH/NIAID On the resistance of Staphylococcus aureus to neutrophil microbicides
Global changes in Staphylococcus aureus gene expression in human blood provide insight into mechanisms of immune evasion and virulence
Gene expression of innate immune response in endothelial cells
HUVECs infected with Staphylococcus aureus isolates from blood and anterior nares
PubMed Similar studies GEO Profiles Analyze DataSet
Staphylococcus aureus induces a muted host response in human blood that blunts the recruitment of neutrophils
Global transcriptome analysis of Staphylococcus aureus biofilms in response to innate immune cells
Transposon sequencing during experimental osteomyelitis reveals hypoxic responses as key components of the staphylococcal-host interaction
SaeRS-dependent inhibition of biofilm formation in Staphylococcus aureus Newman
PubMed Full text in PMC Similar studies SRA Run Selector
Activity of SigB Modulates Virulence Gene Expression in a Murine Staphylococcus aureus Infection Model but Does Not Influence the Host Kidney Gene Expression
Panton-Valentine Leukocidin Does Not Impact Virulence of Community-associated MRSA
Gene expression data from S. aureus-exposed neutrophils
The novel peptide ScrA acts through the SaeRS two component system to regulate virulence gene expression in Staphylococcus aureus
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