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Genome-wide transcriptional analyses of islet-specific CD4+ T cells identify Idd9 genes controlling diabetogenic T cell function
PubMed Full text in PMC Similar studies Analyze with GEO2R
Comparison of gene expression in NOD versus NOD.NOR-Chr4 (NR4) splenic B cells.
Non-obese diabetic (NOD) and disease-resistant NOD.NOR-Chr4 models: splenic B cells
PubMed Full text in PMC Similar studies GEO Profiles Analyze DataSet
NOD model of type 1 diabetes
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EOSS002A
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Type 1 diabetes gene expression profiling
PubMed Similar studies GEO Profiles Analyze DataSet
Interferon-γ-dependent regulatory circuits in immune inflammation highlighted in diabetes
Time course of diabetes-induced, anti-Interferon-γ injected NOD.Scid mice: pancreatic islets
Transcriptome and Molecular Pathways Analysis of CD4 T-Cells from Young NOD Mice
Nonobese diabetic NOD spleen CD4 T-cells: 4-week old females
Nonobese diabetic NOD spleen CD4 T-cells: 3-week old females
Nonobese diabetic NOD spleen CD4 T-cells: 2-week old females
Examination of inflammatory transcripts during a transfer model of type I diabetes.
Diabetogenic BDC T-cell transfer model of type I diabetes: pancreatic islets
Diabetogenic 3A9 T-cell transfer model of type I diabetes: pancreatic islets
Over expression of mRNA for multiple genes including insulin in the PLN of NOD is associated with Islet Autoimmunity
NOD mice positive for Early Insulin AutoAntibodies: pancreatic lymph nodes
Comparison of gene expression signatures in pancreatic islets and pathogenic T lymphocytes from sublines of non-obese diabetic (NOD) mice
The Position Beta57 of IAg7 Controls the Early Anti-Insulin Response and Onset of Diabetes in NOD mice to Link MHC and Disease
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The Position Beta57 of IAg7 Controls the Early Anti-Insulin Response and Onset of Diabetes in NOD mice to Link MHC and Disease [sequencing]
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