Similar studies for GEO DataSets (Select 200151635) - GEO DataSets

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Items: 3

Select item 2001516351.

Gene expression data from T47D human breast ductal carcinoma cells treated with prolactin and/or NIM811

(Submitter supplied) The hormone prolactin is implicated in the pathogenesis of breast cancer, and a subset of prolactin-induced gene expression is mediated by CypA activity. The non-immunosuppressive prolyl isomerase inhibitor, NIM811, is an inhibitor of CypA. We used NIM811 in combination with prolactin treatment in T47D cells to determine differential expression under these conditions.

Organism:: Homo sapiens

Type:: Expression profiling by array

Platform:: GPL571
12 Samples

Download data: CEL

Series

Accession:: GSE151635

ID:: 200151635

PubMed Full text in PMC Similar studies Analyze with GEO2R

Select item 2000155052.

Expression of cyclophilin B is associated with malignant progression and regulation of genes

(Submitter supplied) Microarray was conducted on Illumina Human Ref-6 Version 2 Expression Chip (Illumina, San Diego, CA). Three independent cultures were used for RNA isolation with RNeasy plus mini kit (Qiagen, Valencia, CA). RNA quality was checked by Agilent Bioanalyzer (Agilent, Santa Clara, CA). An Ambion labeling kit was used for labeling cDNA followed by hybridization to Illumina chips. ChIP scan data was extracted by Illumina Beadstudio and subsequently analyzed using Bioconductor lumi package. more...

Organism:: Homo sapiens

Type:: Expression profiling by array

Platform:: GPL6102
12 Samples

Download data

Series

Accession:: GSE15505

ID:: 200015505

PubMed Full text in PMC Similar studies Analyze with GEO2R

Select item 2000996433.

The RhoA guanine nucleotide exchange factor Net1 is required for PyMT mediated mammary gland tumorigenesis and metastasis

(Submitter supplied) The Net1 RhoGEF has been implicated in invasive behavior in vitro, but its impact on tumorigenesis and metastasis in vivo has not yet been established. We developed Net1 knockout mice and profiled the transcriptomes of the resulting tumors to assess impact on pathway activation.