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Deciphering cell states and genealogies of human hematopoiesis with single-cell multi-omics [Extended donors]
PubMed Full text in PMC Similar studies
Deciphering cell states and genealogies of human hematopoiesis with single-cell multi-omics [Crispr_Mouse_Batch2]
Deciphering cell states and genealogies of human hematopoiesis with single-cell multi-omics [Crispr_Mouse_Batch1]
Deciphering cell states and genealogies of human hematopoiesis with single-cell multi-omics [Young2]
Deciphering cell states and genealogies of human hematopoiesis with single-cell multi-omics [Young1_T2]
Deciphering cell states and genealogies of human hematopoiesis with single-cell multi-omics [Young 1]
Deciphering cell states and genealogies of human hematopoiesis with single-cell multi-omics [Aged 2]
Deciphering cell states and genealogies of human hematopoiesis with single-cell multi-omics
Deciphering cell states and genealogies of human hematopoiesis with single-cell multi-omics [Aged 1]
Clonal analysis of lineage fate in unperturbed hematopoiesis
PubMed Full text in PMC Similar studies SRA Run Selector
Simultaneous lineage and transcriptome analysis of hematopoietic stem cell fates
Epigenetic Memory Underlies Cell Autonomous Heterogeneous Behavior of Hematopoietic Stem Cells
PubMed Similar studies SRA Run Selector
Mapping genotypes to chromatin accessibility profiles in single cells
PubMed Similar studies
Mapping genotypes to chromatin accessibility profiles in single cells [Genotyping GoT-ChA amplicon]
Mapping genotypes to chromatin accessibility profiles in single cells [scATAC-seq; Pt01-19;Pt-02 DOGMAseq]
Resolving fate and transcriptome of hematopoietic stem cell clones
Resolving fate and transcriptome of hematopoietic stem cell clones [LT_ST_HSC]
Resolving fate and transcriptome of hematopoietic stem cell clones [multiple cell types]
An Epigenetic Component of Hematopoietic Stem Cell Aging Amenable to Reprogramming Into a Young State
PubMed Full text in PMC Similar studies Analyze with GEO2R
Accumulating mitochondrial DNA mutations drive premature hematopoietic aging phenotypes molecularly distinct from physiologic stem cell aging
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