Similar studies for GEO DataSets (Select 5824) - GEO DataSets

Warning: The NCBI web site requires JavaScript to function. more...

Format

Summary
Summary (text)
Unique Identifier List

Sort by

Default order
Number of Samples (High to Low)
Number of Samples (Low to High)

Send to:

Choose Destination

File
Clipboard
Collections

Links from GEO DataSets

Items: 3

Select item 58241.

Short-term high fat diet effect on epididymal adipose fractions: time course

Analysis of primary adipocytes from epididymis of C57BL/6J males fed a 60% high fat diet (HFD) for up to 7 days to induce obesity. Results provide insight into the molecular mechanisms underlying insulin resistance in adipose tissues during early obesity.

Organism:: Mus musculus

Type:: Expression profiling by array, count, 2 protocol, 3 time, 2 tissue sets

Platform:: GPL6246
Series:: GSE65557
18 Samples

Download data: CEL

DataSet

Accession:: GDS5824

ID:: 5824

PubMed Full text in PMC Similar studies GEO Profiles Analyze DataSet

Select item 2000655572.

Microarray analysis of Adipose tissue of Short-term HFD-fed mice

(Submitter supplied) Tissue inflammation is a key factor underlying insulin resistance in established obesity. Several models of immuno-compromised mice are protected from obesity-induced insulin resistance. However, it is unanswered whether inflammation triggers systemic insulin resistance or vice versa in obesity. The purpose of this study was to assess these questions.

Organism:: Mus musculus

Type:: Expression profiling by array

Dataset:: GDS5824
Platform:: GPL6246
18 Samples

Download data: CEL

Series

Accession:: GSE65557

ID:: 200065557

PubMed Full text in PMC Similar studies Analyze with GEO2R

Select item 2000811933.

Characterising the transcriptional profile of murine 3T3-L1 adipocytes with altered expression of IRF3.

(Submitter supplied) The chronic inflammatory state that accompanies obesity is a major contributor to insulin resistance and other metabolic dysfunction features. Despite recent advances in our understanding of the cellular and secreted factors that promote the inflammatory milieu of obesity, we have much less insight into the transcriptional pathways that drive these processes. While most attention has focused on the canonical inflammatory transcription factor NF-KB, other potentially important factors exist, including members of the interferon regultory factor (IRF) family. more...