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Items: 3

1.

Drosophila CNS mitochondrial dysfunction with ATF4 inhibition RNA-Seq

(Submitter supplied) Mitochondrial dysfunction causes biophysical, metabolic and signalling changes that alter homeostasis and reprogram cells. We used a Drosophila model in which TFAM is overexpressed in the nervous system with or without knock-down of the ER UPR transcription factor ATF4, to investigate the how mitochondrial dysfunction and ER stress signalling affect the transcriptome.
Organism:
Drosophila melanogaster
Type:
Expression profiling by high throughput sequencing
Platform:
GPL19132
16 Samples
Download data: TXT, XLSX
Series
Accession:
GSE128244
ID:
200128244

PubMed Full text in PMC Similar studies SRA Run Selector

2.

Illumina NextSeq 500 (Drosophila melanogaster)

Organism:
Drosophila melanogaster
486 Series
11735 Samples
Download data
Platform
Accession:
GPL19132
ID:
100019132

3.

ATF4 overexpression rep 2

Organism:
Drosophila melanogaster
Source name:
Central nervous system
Platform:
GPL19132
Series:
GSE128244
Download data: TXT
Sample
Accession:
GSM4038309
ID:
304038309

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db=gds|term=GSM4038309[Accession]|query=3|qty=2|blobid=MCID_67292f5e91da88433d1d9cbb|ismultiple=true|min_list=5|max_list=20|def_tree=20|def_list=|def_view=|url=/Taxonomy/backend/subset.cgi?|trace_url=/stat?
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