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Items: 4

1.

Expression profiling of medulloblastoma

(Submitter supplied) Little is known about the genetic regulation of medulloblastoma dissemination, but metastatic medulloblastoma is highly associated with poor outcome. We obtained expression profiles of 23 primary medulloblastomas clinically designated as either metastatic (M+) or non-metastatic (M0) and identified 85 genes whose expression differed significantly between classes. Using a class prediction algorithm based on these genes and a leave-one-out approach, we assigned sample class to these tumors (M+ or M0) with 72% accuracy and to four additional independent tumors with 100% accuracy. more...
Organism:
Homo sapiens
Type:
Expression profiling by array
Dataset:
GDS232
Platform:
GPL74
23 Samples
Download data: CEL
Series
Accession:
GSE468
ID:
200000468
2.
Full record GDS232

Medulloblastoma metastasis

Identification of genes causing medulloblastoma tumors to metastasize. Analyzed 23 primary medulloblastomas clinically designated as either metastatic or non-metastatic.
Organism:
Homo sapiens
Type:
Expression profiling by array, count, 2 disease state sets
Platform:
GPL74
Series:
GSE468
23 Samples
Download data: CEL
DataSet
Accession:
GDS232
ID:
232
3.

[HC_G110] Affymetrix Human Cancer Array

(Submitter supplied) Affymetrix submissions are typically submitted to GEO using the GEOarchive method described at http://www.ncbi.nlm.nih.gov/projects/geo/info/geo_affy.html Has 2059 entries and was indexed 29-Jan-2002. The Human Cancer G110 array enables focused and cost-effective expression studies in cancer biology. A panel of leading cancer researchers selected the specific set of 1,700 full-length human genes implicated in cancer that comprise this array. more...
Organism:
Homo sapiens
1 DataSet
1 Series
23 Samples
Download data
Platform
Accession:
GPL74
ID:
100000074
4.

KPeterson-NonMet-25G110-s2

Organism:
Homo sapiens
Source name:
Human brain tumor
Platform:
GPL74
Series:
GSE468
Dataset:
GDS232
Download data: CEL
Sample
Accession:
GSM4331
ID:
300004331
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Supplemental Content

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