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Quantitative whole transcriptomics sequencing of progeria-derived cells point to a key role of nucleotide metabolism in premature aging
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Comparison of Hutchinson–Gilford Progeria Syndrome fibroblast cell lines to control fibroblast cell lines
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Hutchinson-Gilford progeria syndrome: fibroblast (HG-U133B)
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Hutchinson-Gilford progeria syndrome: fibroblast (HG-U133A)
Epigenetic deregulation of lamina-associated domains in Hutchinson-Gilford Progeria Syndrome
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Epigenetic deregulation of lamina-associated domains in Hutchinson-Gilford Progeria Syndrome (RNA-Seq)
Epigenetic deregulation of lamina-associated domains in Hutchinson-Gilford Progeria Syndrome (ATAC-Seq)
PubMed Full text in PMC Similar studies SRA Run Selector
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Bone dysplasia in Hutchinson-Gilford Progeria Syndrome is associated with dysregulated differentiation and function of bone cell populations.
Recapitulation of human premature aging by using iPSCs from Hutchinson-Gilford progeria syndrome
Induced pluripotent stem cell-based accelerated aging model
PubMed Full text in PMC Similar studies GEO Profiles Analyze DataSet
Reprogramming Hutchinson-Gilford Progeria Syndrome fibroblasts resets epigenomic landscape in patient-derived induced pluripotent stem cells [ChIP-Seq]
PML2-mediated thread-like nuclear bodies mark late senescence in Hutchinson–Gilford progeria syndrome
PML2‐mediated thread‐like nuclear bodies mark late senescence in Hutchinson–Gilford progeria syndrome [ChIP-seq]
PML2‐mediated thread‐like nuclear bodies mark late senescence in Hutchinson–Gilford progeria syndrome [RNA-seq]
Endothelial and systemic upregulation of miR-34a-5p fine-tunes senescence in progeria
Lmnadelta9 mouse gene expression study
Lamin A exon 9 deletion effect on fibroblasts
BRD4 binding sites in transformed fibroblasts
Expression data from transformed WT and HGPS cell lines, including HGPS cells after knock-down of BRD4
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