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Copy-number profiling by SNP array of 4 acute myeloid leukemia (AML) cell lines
PubMed Similar studies
Transcriptome profiling by RNA-sequencing of the AML cell lines UCSD-AML1 and ELF-153.
PubMed Similar studies Analyze with GEO2RSRA Run Selector
Gene expression data of serially sorted primary AML patient blasts prior and after treatment with the DNMT inhibitor decitabine (DAC)
PubMed Similar studies Analyze with GEO2R
Methylation data of serially sorted primary AML patient blasts prior and after treatment with the DNMT inhibitor decitabine (DAC)
PubMed Full text in PMC Similar studies
Assay for Transposase Accessible Chromatin (ATAC)-sequencing of the AML cell line U937
Transcriptome profiling by RNA-sequencing of the AML cell line U937.
PubMed Full text in PMC Similar studies Analyze with GEO2RSRA Run Selector
Transcriptome profiling by RNA-sequencing of the AML cell line MOLM-13.
The Effect of Decitabine and ATRA on the Methylome in the AML cell line U-937
PubMed Full text in PMC Similar studies Analyze with GEO2R
Multi-omics analysis of myelodysplastic syndromes resistance to epigenetics threrary reveals PI3K/Akt activation mediated by epigenetic silencing of PTEN and epi-transcriptional silencing of MDM2
Multi-omics analysis of myelodysplastic syndromes resistance to epigenetics threrary reveals PI3K/Akt activation mediated by epigenetic silencing of PTEN and epi-transcriptional silencing of MDM2 [transcriptome]
Multi-omics analysis of myelodysplastic syndromes resistance to epigenetics threrary reveals PI3K/Akt activation mediated by epigenetic silencing of PTEN and epi-transcriptional silencing of MDM2 [methylome]
Multi-omics analysis of myelodysplastic syndromes resistance to epigenetics threrary reveals PI3K/Akt activation mediated by epigenetic silencing of PTEN and epi-transcriptional silencing of MDM2 [epitranscriptome]
PubMed Full text in PMC Similar studies SRA Run Selector
A Multicenter, First-in-Pediatrics, Randomized Study to Evaluate the Safety and Efficacy of Decitabine as Epigenetic Priming with Induction Chemotherapy in Children with Acute Myelogenous Leukemia
Analysis of genome-wide methylation and gene expression induced by decitabine treatment in HL60 leukemia cell line
Mitotic perturbation is a key mechanism of action of decitabine in myeloid tumor treatment II
Mitotic perturbation is a key mechanism of action of decitabine in myeloid tumor treatment
Endogenous retroviruses are a source of oncogenic enhancers in acute myeloid leukemia
Endogenous retroviruses are a source of oncogenic enhancers in acute myeloid leukemia [RNA-Seq]
Endogenous retroviruses are a source of oncogenic enhancers in acute myeloid leukemia [DNase-Seq]
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