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Non-obese diabetic (NOD) and disease-resistant NOD.NOR-Chr4 models: splenic B cells
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Comparison of gene expression in NOD versus NOD.NOR-Chr4 (NR4) splenic B cells.
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Genome-wide transcriptional analyses of islet-specific CD4+ T cells identify Idd9 genes controlling diabetogenic T cell function
Deletion of Vβ3+CD4+ T cells by endogenous mouse mammary tumor virus 3 prevents type 1 diabetes induction by autoreactive CD8+ T cells
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The Position Beta57 of IAg7 Controls the Early Anti-Insulin Response and Onset of Diabetes in NOD mice to Link MHC and Disease
The Position Beta57 of IAg7 Controls the Early Anti-Insulin Response and Onset of Diabetes in NOD mice to Link MHC and Disease [sequencing]
PubMed Full text in PMC Similar studies SRA Run Selector
The Position Beta57 of IAg7 Controls the Early Anti-Insulin Response and Onset of Diabetes in NOD mice to Link MHC and Disease [RT-PCR]
Comparison of gene expression signatures in pancreatic islets and pathogenic T lymphocytes from sublines of non-obese diabetic (NOD) mice
Transcriptome and Molecular Pathways Analysis of CD4 T-Cells from Young NOD Mice
Nonobese diabetic NOD spleen CD4 T-cells: 4-week old females
Nonobese diabetic NOD spleen CD4 T-cells: 3-week old females
Nonobese diabetic NOD spleen CD4 T-cells: 2-week old females
Genetic and transcriptome analyses of early T-cell checkpoint failure and leukemia initiation in Rag1-deficient NOD mice
Comparison of gene expression between islets from NOD-Rag and NOD-Idd22 mice
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