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Items: 3

1.

Role of JunB in Th17 cell effector stability

(Submitter supplied) This SuperSeries is composed of the SubSeries listed below.
Organism:
Mus musculus
Type:
Expression profiling by high throughput sequencing; Genome binding/occupancy profiling by high throughput sequencing
Platforms:
GPL21103 GPL13112
14 Samples
Download data: XLS
Series
Accession:
GSE98414
ID:
200098414

PubMed Full text in PMC Similar studies

2.

Role of JunB in Th17 cell effector stability [RNA-seq]

(Submitter supplied) Here we identify the activator protein-1 (AP-1) factor JunB as an essential regulator of Th17 cell identity. JunB activates the expression of Th17 lineage-specifying genes, and coordinately represses genes controlling Th1 and Treg fate. Through regulatory analysis, we find that JunB is a core regulator of global transcriptional programs that promote Th17 cell identity and restrict alternative CD4+ T cell potential.
Organism:
Mus musculus
Type:
Expression profiling by high throughput sequencing
Platform:
GPL13112
4 Samples
Download data: TAB
Series
Accession:
GSE98413
ID:
200098413

PubMed Full text in PMC Similar studies SRA Run Selector

3.

Role of JunB in Th17 cell effector stability [ChIP-seq]

(Submitter supplied) Here we identify the activator protein-1 (AP-1) factor JunB as an essential regulator of Th17 cell identity. JunB activates the expression of Th17 lineage-specifying genes, and coordinately represses genes controlling Th1 and Treg fate. Through regulatory analysis, we find that JunB is a core regulator of global transcriptional programs that promote Th17 cell identity and restrict alternative CD4+ T cell potential.
Organism:
Mus musculus
Type:
Genome binding/occupancy profiling by high throughput sequencing
Platforms:
GPL21103 GPL13112
10 Samples
Download data: XLS
Series
Accession:
GSE98412
ID:
200098412

PubMed Full text in PMC Similar studies SRA Run Selector

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