Global transcriptomic response to chilling temperature in Col-0 wild type and sig5-3 (SALK_141383C) mutant Arabidopsis plants.

Prognostic factors of atypical meningiomas treated with gross total resection and early radiotherapy: microRNA-221/222 co-modulated radiosensitivity regulatory mechanism and malignent progression

Topological isolation as a mechanism for precise control of developmental regulators in mammalian genomes [RNA-seq]

Topological isolation as a mechanism for precise control of developmental regulators in mammalian genomes

Topological isolation as a mechanism for precise control of developmental regulators in mammalian genomes [HiC]

Single-cell analysis of whole hepatic non-parenchymal cells from normal and CCl4-injured mice

Gene expression data from memory Rictor-/- and WT Tfh and Th1 cells in LCMV acute infection

Durable Suppression of Acquired MEK Inhibitor Resistance in Cancer by Sequestering MEK from ERK and Promoting Anti-Tumor T-cell Immunity [RNA-seq]

Durable Suppression of Acquired MEK Inhibitor Resistance in Cancer by Sequestering MEK from ERK and Promoting Anti-Tumor T-cell Immunity [TCR-seq]

Durable Suppression of Acquired MEK Inhibitor Resistance in Cancer by Sequestering MEK from ERK and Promoting Anti-Tumor T-cell Immunity [single-cell RNA-seq]

Durable Suppression of Acquired MEK Inhibitor Resistance in Cancer by Sequestering MEK from ERK and Promoting Anti-Tumor T-cell Immunity

STAG proteins mediate heterochromatin organization to support translation and cell identity [RNA-Seq]

STAG proteins mediate heterochromatin organization to support translation and cell identity [UMI-4C]

STAG proteins mediate heterochromatin organization to support translation and cell identity [Iso-Seq]

PEZIC_STAG1_Pluripotency

ARID1A deficiency promotes pancreatic tumorigenesis by reducing KRAS-induced senescence (ATAC-Seq)

ARID1A deficiency promotes pancreatic tumorigenesis by reducing KRAS-induced senescence (RNA-Seq I)

ARID1A deficiency promotes pancreatic tumorigenesis by reducing KRAS-induced senescence (RNA-Seq II)

Topological isolation as a mechanism for precise control of developmental regulators in mammalian genomes [RNA-seq II]

Topological isolation as a mechanism for precise control of developmental regulators in mammalian genomes (Hi-C II)